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Yingzhi Huang

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Articles

Learning-Based Formation Control of Distributed MASs With Actuator Hysteresis Using Graph Convolutional Networks

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By Jiaqi Liu, Ye Zhang, Yingzhi Huang
| Wiley Online Library Verified
Conflicts of Interest The authors declare no conflicts of interest. Data Availability Statement The data that support the findings of this study are available from the corresponding author upon reasonable request. References 1, , , , , and , “A Survey of an Intelligent Multi-Agent Formation Control,” Applied Sciences 13, no. 10 (2023): 5934, https://doi.org/10.3390/app13105934.
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Transcriptomic and Metabolomic Signatures of Acute Circadian Misalignment in Mice

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By Yingzhi Huang, Hui Wang, Haonan Li, Baoyin Ren
| Wiley Online Library Verified
Conflicts of Interest The authors declare no conflicts of interest. Supporting Information Filename Description jpi70075-sup-0001-Supplementary_Data_3-LC-MS_Analysis.docx15.9 KB Supplementary_Data_3-LC-MS_Analysis. jpi70075-sup-0002-Supplementary_Data1-dryR.xlsx7.3 MB Supplementary_Data1-dryR. jpi70075-sup-0003-Supplementary_Data2.docx146.1 KB Supplementary_Data2. jpi70075-sup-0004-Supplementary_Figure.pptx6.1 MB Supplementary_Figure. jpi70075-sup-0005-Table.pptx35.7 KB Table.
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Germline intergenic duplications at Xq26.1 underlie Bazex-Dupré-Christol basal cell carcinoma susceptibility syndrome

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By Anette Bygum, Yanshan Liu, Siddharth Banka, Yingzhi Huang
| Wiley Online Library Verified
Abstract Background Bazex–Dupré–Christol syndrome (BDCS; MIM301845) is a rare X-linked dominant genodermatosis characterized by follicular atrophoderma, congenital hypotrichosis and multiple basal cell carcinomas (BCCs). Previous studies have linked BDCS to an 11·4-Mb interval on chromosome Xq25-q27.1. However, the genetic mechanism of BDCS remains an open question. Objectives To investigate the genetic aetiology and molecular mechanisms underlying BDCS.
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