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Yining Lu on Muck Rack

Yining Lu

As seen in: Wiley Online Library, arXiv, MDPI, SAGE Publishing, Taylor & Francis Online, ACS Publications, Dove Medical Press, The Journal of Rheumatology, Arthroscopy: the Journal of Arthroscopic & Related Surgery, Arthroscopy, Sports Medicine, and Rehabilitation
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Articles

Capsular Contributions to Hip Joint Stability: Clinical Anatomy and Implications for Surgical Management

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By Yining Lu, Brandon Cabarcas, rana ahmad
| Wiley Online Library Verified
, , , et al. 2026. “Capsular Contributions to Hip Joint Stability: Clinical Anatomy and Implications for Surgical Management.” Clinical Anatomy1–11. https://doi.org/10.1002/ca.70182. Data Availability Statement Data sharing not applicable to this article as no datasets were generated or analysed during the current study. References , , and . 2022. “Capsule Closure of Periportal Capsulotomy for Hip Arthroscopy.” Arthroscopy Techniques 11, no. 6: e1117–e1122. https://doi.org/10.1016/j.eats.2022.02.018.
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Comparing Novel and Legacy Health Assessment Questionnaire Scoring Methods: A Scleroderma Patient-centered Intervention Network Cohort Study

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By Yining Lu, Daphna Harel, Klint Kanopka
| The Journal of Rheumatology Verified
Key Indexing Terms: disability Health Assessment Questionnaire item response trees scleroderma systemic sclerosis Systemic sclerosis (SSc), also known as scleroderma, is a rare, complex autoimmune rheumatic disease characterized by fibrosis of the skin and multiple internal organ systems, including the lungs, heart, kidneys, musculoskeletal system, and gastrointestinal (GI) tract.1,2 Factors that may be associated with disability in SSc include fatigue, dyspnea, pain and pruritus, GI...
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Sialidase Neu3 Induced Sialic Acid Disorder and Promoted Vascular Endothelial Injury Through Transcription Factor SP3

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By Yining Lu, Peng Xiang, Qingqiu Chen
| Wiley Online Library Verified
1 Introduction Sialic acid, a nine-carbon monosaccharide with a negative charge, has recently garnered attention as a significant marker of cardiovascular events, with N-acetylneuraminic acid (Neu5Ac) being the predominant form in vivo [1]. Previous studies have demonstrated its critical role in promoting the progression of coronary artery diseases by activating the Rho/ROCK-JNK/ERK pathway in vitro and in vivo [2].
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