Yinqi Zhao

AAV9 Adeno-associated virus 9 CK Creatine kinase H/R Hypoxia/reoxygenation IHD Ischemic heart disease ISG15 Interferon-stimulated gene 15 LDH Lactate dehydrogenase MI Myocardial infarction MI/RI Myocardial ischemia-reperfusion injury NRCMs Neonatal rat cardiomyocytes PCI Percutaneous coronary intervention ROS Reactive oxygen species SR-A3 Scavenger receptor class A3 STING Stimulator of interferon response cGAMP interactor TUNEL Terminal deoxynucleotidyl transferase-mediated dUTP Nick-end...

Abstract Background: Alveolar echinococcosis (AE), caused by the larval stage of Echinococcus multilocularis, exhibits infiltrative, tumor-like behavior in the liver and persists within its tolerogenic immune environment. Although T cells are central to host defense, the stage-specific remodeling of their lineage states during AE remains unclear. Methods: A secondary AE infection model was established by portal vein injection of approximately 1,000 viable protoscoleces in C57BL/6 mice.

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