Zhi-ying Jiang

1 Introduction Sepsis is capable of inducing organ dysfunction, which typically stems from the host's inadequate immune response to infectious agents [1]. Septic cardiomyopathy (SCM) is an acute myocardial injury mediated by sepsis [2, 3]. Statistical data indicates that the mortality rate of septic patients complicated with myocardial injury is as high as 70%–90% [4]. Despite the extensive research efforts devoted to septic cardiomyopathy, its underlying mechanisms remain incompletely understood.