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Adeline Barthelemy

As seen in: Nature, Cell Press
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Articles

Surface CD81 supports leukemia stem cell function and reveals a therapeutic vulnerability in acute myeloid leukemia

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By Fanny Gonzales, Pauline Peyrouze, Djohana Laurent, Thomas Boyer, Nihad Boukrout, Cyril Couturier, Véronique Lisi, Adeline Barthelemy, François Sevrin, Adriana Plesa, Antonino Bongiovanni, Nicolas Pottier, Claude Preudhomme, Vincent-Philippe Lavallée, Konstantinos Geles, Nicolas Duployez, Céline Berthon, Meyling Cheok, Soizic Houdiard, Christophe Roumier
| Nature Verified
Abstract Relapse remains the leading cause of mortality in acute myeloid leukemia (AML), largely due to the persistence of therapy-resistant leukemia stem cells (LSCs). However, surface determinants that sustain LSC function and disease aggressiveness remain incompletely defined. Here, we identify the tetraspanin CD81 as a regulator of LSC function, progression and treatment resistance in AML.
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Gut Dysbiosis during Influenza Contributes to Pulmonary Pneumococcal Superinfection through Altered Short-Chain Fatty Acid Production

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By Valentin Sencio, Adeline Barthelemy, Daphnée Soulard, Luciana P. Tavares, Marina G. Machado, Céline Cuinat, Celso Martins Queiroz-Junior, Marie-Louise Noordine, Sophie Salomé-Desnoulez, Lucie Deryuter, Benoit Foligné, Céline Wahl, Benoit Frisch
| Cell Press Verified
Highlights • Influenza alters the production of SCFAs by the gut microbiota • The dysbiotic microbiota transfers susceptibility to respiratory bacterial infection • Supplementation with acetate restores the killing activity of alveolar macrophages • Activation of the SCFA receptor FFAR2 protects against bacterial superinfection Summary Secondary bacterial infections often complicate viral respiratory infections.
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Interleukin-22 Immunotherapy during Severe Influenza Enhances Lung Tissue Integrity and Reduces Secondary Bacterial Systemic Invasion

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By Ronan Le Gofficf, Adeline Barthelemy, aUniversitaire de Lille, Valentin Sencio
| American Society for Microbiology (ASM)
Severe bacterial (pneumococcal) infections are commonly associated with influenza and are significant contributors to the excess morbidity and mortality of influenza. Disruption of lung tissue integrity during influenza participates in bacterial pulmonary colonization and dissemination out of the lungs. Interleukin-22 (IL-22) has gained considerable interest in anti-inflammatory and anti-infection immunotherapy over the last decade.
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