Alkmini Damkou on Muck Rack

Alkmini Damkou

As seen in: Nature, Cell Press
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Articles

Apolipoprotein E aggregation in microglia initiates Alzheimer’s disease pathology by seeding β-amyloidosis

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By Seiji Kaji, Stefan A. Berghoff, Lena Spieth, Lennart Schlaphoff, Andrew O. Sasmita, Simona Vitale, Luca Büschgens, Shreeya Kedia, Martin Zirngibl, Taisiia Nazarenko, Alkmini Damkou, Leon Hosang, Constanze Depp, Frits Kamp, Patricia Scholz, David Ewers, Martin Giera, Till Ischebeck, Wolfgang Wurst, Benedikt Wefers, Martina Schifferer, Michael Willem, Klaus-Armin Nave, Christian Haass, Thomas Arzberger, Sarah Jäkel, Oliver Wirths, Gesine Saher, Mikael Simons
| Cell Press Verified
Main text (Immunity 57, 2651–2668.e1–e12; November 12, 2024) The authors noticed that the bar graph shown in Figure 5C showing MX04+ aggregates for TREM2KO cells was generated with the wrong numbers. The panel has now been corrected. The corrected figure panel does not affect the conclusions. We also noticed two minor textual errors that were inadvertent. The legend in Figure 5C did not mention the statistical test for the right graph.
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Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease - Nature

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By Constanze Depp, Ting Sun, Lena Spieth, Wiebke Möbius, Gesine Saher, Hauke B. Werner, Silvia Zampar, Oliver Wirths, Mikael Simons, Takaomi C. Saido, Christian Haass, Daniel H Geschwind, Hannelore Ehrenreich, Alkmini Damkou, Maik Thalmann
| Nature Verified
Abstract The incidence of Alzheimer’s disease (AD), the leading cause of dementia, increases rapidly with age, but why age constitutes the main risk factor is still poorly understood. Brain ageing affects oligodendrocytes and the structural integrity of myelin sheaths1, the latter of which is associated with secondary neuroinflammation2,3.
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