Anamaria Ciubara

You are already at the latest version Background/Objectives: Neuroinflammation is increasingly recognized as a key mechanism linking infectious diseases with psychiatric disorders through interactions between peripheral immune activation, metabolic pathways, and brain network alterations. This review aimed to synthesize current evidence on the neuroimmune mechanisms and biomarkers underlying infection-associated psychiatric disorders.

Introduction Tuberculosis (TB) is one of the oldest known infectious diseases and remains a major public health problem worldwide.1,2 Although significant progress has been made in diagnosis and treatment, TB continues to affect millions of people annually, including a significant number of children. In the modern context of public health, TB is no longer only a medical problem, but also a psychosocial one, profoundly affecting the lives and well-being of patients.

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