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Articles by Anna Jasz
Movement-stabilized three-dimensional optical recordings of membrane potential changes and calcium dynamics in hippocampal CA1 dendrites
Main text (Neuron , ◼◼◼–◼◼◼; May 20, 2026) The authors regret that errors appeared in the originally published manuscript. Figure S1G is correct; however, the corresponding text in the section entitled “3D-RTMC steps” contained errors. Specifically, we used four AOD cards rather than three, and the PMT cards computed the weighted and unweighted sums for the three axes, which were then transferred to and further processed in parallel by the four AOD cards, as shown in detail in Figure S1G.
Movement-stabilized three-dimensional optical recordings of membrane potential changes and calcium dynamics in hippocampal CA1 dendrites
Keywords two-photon voltage imaging dendritic computation backpropagating action potentials 3D real-time motion correction hippocampal CA1 pyramidal neurons voltage-calcium coupling in vivo dendritic imaging acousto-optic deflector soma-dendrite attenuation branch-order-dependent signaling Get full text access Log in, subscribe or purchase for full access. References 1. Stuart, G.J. ∙ Spruston, N. Dendritic integration: 60 years of progress Nat. Neurosci. 2015; 18:1713-1721 2. Major, G.
Persistently increased post-stress activity of paraventricular thalamic neurons is essential for the emergence of stress-induced alterations in behaviour
Loading metrics Open Access Peer-reviewed Research Article Citation: Jász A, Biró L, Buday Z, Király B, Szalárdy O, Horváth K, et al. (2025) Persistently increased post-stress activity of paraventricular thalamic neurons is essential for the emergence of stress-induced alterations in behaviour. PLoS Biol 23(1): e3002962. https://doi.org/10.1371/journal.pbio.3002962 Academic Editor: Heather A.
Persistently increased post-stress activity of paraventricular thalamic neurons is essential for the emergence of stress-induced maladaptive behavior
New Results doi: https://doi.org/10.1101/2023.04.20.537629 Abstract Traumatic events can immediately lead to debilitating symptoms collectively called Acute Stress Disorder (ASD), however the mechanisms of ASD are poorly understood. Using a rodent model of ASD here we identify a crucial communication bottleneck between the brainstem and the forebrain, the calretinin-positive neurons in the paraventricular thalamus (PVT/CR+), that controls ASD.
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