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Anthony Hannan on Muck Rack

Anthony Hannan

Melbourne
As seen in: Nature, Wiley Online Library, Yahoo News Australia, Memory Studies, Frontiers, The Conversation, New Zealand Herald, The Epoch Times, Pursuit, BioRxiv and
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Articles

Psilocybin produces amplified acute responses and sex-specific long-term increases in sensorimotor gating in the mGlu5 knockout mouse model of schizophrenia

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By James Gattuso, Ahmed Kamal, Zoe Phelan, Matthew Hale, Anthony Hannan, Thibault Renoir, Jennyfer M. Payet, Kato Havaux, Jenna Hendey, Bilgenur Bezcioglu, Maddy Orchard, Divyesh Bhanot
| Nature Verified
Abstract Psilocybin is a serotonergic psychedelic drug with emerging therapeutic applications, yet its actions under glutamatergic dysfunction, and relevance to schizophrenia and associated psychiatric disorders, remain unclear. We examined the acute and long-lasting effects of psilocybin (1 mg/kg, intraperitoneal) on behavioral and neural outcomes in the metabotropic glutamate receptor 5 (mGlu5) knockout (KO) mouse model of schizophrenia.
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Paternal cytokine administration alters sperm small ncRNAs and offspring brain and behavior

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By Pamudika Kiridena, Nicholas van de Garde, Carolina Gubert, Sonali N. Reisinger, Anthony Hannan, Bethany A. Masson
| Nature Verified
Abstract Paternal pre-conceptual exposure to pathogenic infection can alter offspring phenotypes via changes to sperm epigenetics, including small non-coding RNAs (ncRNAs). This phenomenon occurs even in the absence of infection when pathogen mimetics trigger paternal immune activation (PIA). While this implicates a shared component of the immune response, the specific mechanisms responsible remain unknown.
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Gut microbiome alterations are sex-dependently associated with brain abnormalities in a mouse model of Neurofibromatosis type I

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By Sonali N. Reisinger, Geraldine Kong, Asim Jamil Muhammad, Da Lu, Carolina Gubert, Gabriel Dabscheck, Jonathan Payne, Anthony Hannan, Nicholas van de Garde, Pranav Adithya, Pamudika Kiridena
| Nature Verified
Abstract Neurofibromatosis type 1 (NF1) is a genetic condition presenting with variable symptomatology, however most individuals will demonstrate cognitive and behavioural difficulties, including autism. Using a heterozygous germline knockout mouse model of NF1 (Nf1 +/-), we performed in-depth behavioural evaluations encompassing learning and memory, stereotypy, social interaction, anxiety- and depression-like behaviour.
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