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Aris Baras

As seen in: Nature, Neurology
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Articles

A Liver-Targeted CIDEB siRNA in Adults with MASLD

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By Thomas Norton, Jorg Taubel, Aris Baras
| nej.md
Notes Thomas D. Norton can be contacted at . * Complete lists of the collaborators from each group are listed in the Supplementary Appendix, available at NEJM.org. Thomas D. Norton and Jorg Taubel, and Aris Baras, Gary A. Herman, and David J. Lederer, contributed equally to this letter. This letter was published on November 8, 2026, at NEJM.org. A data sharing statement provided by the authors is available with the full text of this letter at NEJM.org. Supported by Regeneron Pharmaceuticals.
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FNIP1 variants are associated with favourable metabolism in 1 million humans

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By George Hindy, René Adam, Olukayode Sosina, David Blair, Joseph L. Herman, Joseph Lee, Peter Dornbos, Ernst Mayerhofer, Arthur Gilly, Charleen Hunt, Benjamin J. Geraghty, Karl Landheer, Liron Ganel, Antoine Baldassari, Chuanyi Zhang, Ivory J. Mintah, Daphne S. Sun, Angel Coppola, Kyle Brown, Tram Nguyen, Xin Xin Xia, Ghs-rgc DiscovEhr Collaboration, Penn Medicine Biobank, Malmö Diet, Daniel Rader, Olle Melander, Christopher D. Still, Jaime Berumen, Pablo Kuri-Morales, Jesus Alegre-Díaz, Jason Torres, Jonathan Emberson, Rory Collins, Roberto Tapia-Conyer, Suganthi Balasubramanian, Marcus Jones, Michelle LeBlanc, michelle leblanc, Andrew Murphy Verified, Christos A. Kyratsous, John D. Overton, Jeffrey Reid, Goncalo Abecasis, Jonathan Marchini, Cristen J. Willer, George D. Yancopoulos, Mark W. Sleeman, Jonas Bovijn, Adam E. Locke, Aris Baras, Niek Verweij, Viktoria Gusarova, Luca A. Lotta, Dwaine Pryce, UCLA-RGC ATLAS Collaboration, UTSW-Dallas Biobank
| Nature Verified
Abstract Altered energy metabolism is a shared driver across cardiometabolic diseases—the leading cause of death globally1. Energy metabolism varies between individuals and is partly heritable2,3,4,5,6,7,8,9.
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Polygenic risk score and 20-year prostate cancer-specific mortality and survival

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By Anna Plym, Anqi Wang, Konrad H. Stopsack, Yiwen Zhang, Aris Baras, Isabel Drake, Mark Clements Verified, Kathryn L. Penney, Edward L. Giovannucci, Lorelei A. Mucci
| Nature Verified
Abstract Polygenic risk scores (PRSs) strongly discriminate for prostate cancer risk at the population level. The role of these genetic scores in determining prostate cancer survival is unclear, potentially due to methodological issues. We included 19,607 men from the Malmö Diet and Cancer Study (MDCS) and the Health Professionals Follow-up Study (HPFS) and analyzed 20-year incidence and mortality (from full cohort analyses) and survival (from case-only analyses) according to a 451-variant PRS.
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