Avenue Hubert Curien on Muck Rack

Avenue Hubert Curien

As seen in: Cell Press
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Articles

MCM8- and MCM9 Deficiencies Cause Lifelong Increased Hematopoietic DNA Damage Driving p53-Dependent Myeloid Tumors

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By Malik Lutzmann, Avenue Hubert Curien, Florence Bernex, Dana Hodroj
| Cell Press
Highlights • MCM8−/− and MCM9−/− mice show lifelong increased DNA damage in their bone marrow • The mice develop myeloid tumors resembling human myelodysplastic syndrome as they age • Loss of RB-mediated cell cycle control allows myeloid tumor proliferation • Additional loss of TP53 switches tumor development to aggressive T cell lymphoma Summary Hematopoiesis is particularly sensitive to DNA damage. Myeloid tumor incidence increases in patients with DNA repair defects and after chemotherapy.
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