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Barbara De Moerloose on Muck Rack

Barbara De Moerloose

As seen in: Nature
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Articles

Age-dependent phenotypic and molecular evolution of pediatric MDS arising from GATA2 deficiency - Blood Cancer Journal

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By Lili Kotmayer, Guolian Kang, Ayami Yoshimi, Rebecca Voss, Barbara De Moerloose, Henrik Hasle, Kirsi Jahnukainen, Riccardo Masetti, Jochen Buechner, Marek Ussowicz, Paula Kjöllerström, Dominik Turkiewicz, Markus Schmugge, Victoria Okhomina, Cristian Sotomayor, Claudia Wehr, Nathan Gray, Alessandra Giorgetti, Charlotte M. Niemeyer, Luca Vinci
| Nature Verified
Abstract GATA2 deficiency is an autosomal dominant transcriptopathy disorder with high risk for myelodysplastic syndrome (MDS). To elucidate genotype-phenotype associations and identify new genetic risk factors for MDS, we analyzed 218 individuals with germline heterozygous GATA2 variants. We observed striking age-dependent incidence patterns in GATA2-related MDS (GATA2-MDS), with MDS being absent in infants, rare before age 6 years, and steeply increasing in older children.
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Management of children and adolescents with chronic myeloid leukemia in chronic phase: International pediatric chronic myeloid leukemia expert panel recommendations | Leukemia

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By Frédéric Millot, Nirmalya Roy Moulik, Sanjay Tewari, Mahmoud A Hammad, Herbert Pichler, Haruko Shima, Wenyu Yang, Roula Farah, Barbara De Moerloose, Kirk R. Schultz, Krzysztof Kalwak, Nobuko Hijiya, Markus Metzler
| Nature Verified
Abstract The treatment strategy for children and adolescents with chronic myeloid leukemia in the chronic phase (CML-CP) has evolved from allogeneic hematopoietic stem cell transplantation (HSCT) to tyrosine kinase inhibitors (TKIs).
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Second allogeneic stem cell transplantation can rescue a significant proportion of patients with JMML relapsing after first allograft - Bone Marrow Transplantation

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By Barbara De Moerloose, Henrik Hasle, Tayfun Güngör, Jochen Buechner, Marco Zecca, Peter Lang, Roland Meisel, Herbert Pichler, Franco Locatelli, Brigitte Strahm, Charlotte M. Niemeyer, Peter Noellke
| Nature Verified
To the Editor: Juvenile myelomonocytic leukemia (JMML) is a rare myeloproliferative disease of early childhood [1]. More than 90% of patients harbor mutations in PTPN11, KRAS, NRAS, CBL, or NF1. For most patients, allogeneic hematopoietic stem cell transplantation (HSCT) is the only curative therapy, while relapse is the major cause of treatment failure recorded in about 35% of patients [2, 3].
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