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Author Correction: Neuroimmune cardiovascular interfaces control atherosclerosis
Icon version of the Flipboard logo nature.com - Sarajo K. Mohanta , Li Peng, Yuanfang Li, Shu Lu, Ting Sun, Lorenzo Carnevale, Marialuisa Perrotta, Zhe Ma, Benjamin Förstera, Karen Stanic, Chuankai Zhang, Xi Zhang, Piotr Szczepaniak, Mariaelvy Bianchini, Borhan R. Saeed, Raimondo Carnevale, Desheng Hu, Ryszard Nosalski, Fabio Pallante, Michael Beer, Donato Santovito, Ali Ertürk, Thomas C. Mettenleiter, Barbara G. Klupp, Remco T. A.
Viruses | Free Full-Text | Mass-Spectrometric Evaluation of the African Swine Fever Virus-Induced Host Shutoff Using Dynamic Stable Isotope Labeling with Amino Acids in Cell Culture (SILAC)
3.2. Quantitative Evaluation of the ASFV-Induced Vhs Following the validation of the experimental setup, the ASFV-induced vhs was evaluated. As for PrV, infections with ASFV were verified by immunoblot (Figure S1C) and by mass-spectrometric detection of 122 ASFV proteins (Supplementary Table S1) in the cell lysates.
In Vitro Viral Evolution Identifies a Critical Residue in the Alphaherpesvirus Fusion Glycoprotein B Ectodomain That Controls gH/gL-Independent Entry
Research Article Melina Vallbracht, Henriette Lötzsch, Barbara G. Klupp, Walter Fuchs, Benjamin Vollmer, Kay Grünewald, Marija Backovic, Felix A. Rey, Thomas C. Mettenleiter Anne Moscona, Editor DOI: 10.1128/mBio.00557-21 ABSTRACT Herpesvirus entry and spread requires fusion of viral and host cell membranes, which is mediated by the conserved surface glycoprotein B (gB).
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