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Barbara Schrörs

As seen in: Nature, BioRxiv
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Articles

The MYB-related transcription factor MYPOP acts as a selective regulator of cancer cell growth

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By Johannes Strunk, Sascha Wagner, Kirsten Freitag, Tatjana Döring, Tobias Bopp, Matthias Klein, Krishnaraj Rajalingam, Gregory Harms, Federico Marini, Carina Osterhof, Marc Schneider, Alina Henrich, Martin Suchan, Barbara Schrörs, Mustafa Diken, Alena Hüppner, Mahwish Sial, Matthias Plath, Mika B. Sheriff, Annekathrin S. Nedwed, Thomas Hankeln, Andrea Nubbemeyer, Mario Perkovic, Sebastian Kreiter, Anne Kölsch, Luise Florin
| Nature Verified
Abstract The MYB-related transcription factor and partner of profilin (MYPOP or p42POP) is a ubiquitously expressed yet understudied protein recently identified as a restriction factor of oncogenic human papillomaviruses (HPV) and proposed tumor suppressor. Here we show that in HPV-transformed cervical cancer cells, MYPOP induces alterations in cell morphology, silences viral and cellular oncogenes including E6 and MYC, and stimulates the release of the cancer-killing cytokine interleukin-24.
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Author Correction: Identification of neoantigens for individualized therapeutic cancer vaccines - Nature Reviews Drug Discovery

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By Barbara Schrörs
| Nature Verified
Correction to: Nature Reviews Drug Discovery https://doi.org/10.1038/s41573-021-00387-y, published online 1 February 2022. In the version of the article initially published, one of the affiliations for Franziska Lang (Faculty of Biology, Johannes Gutenberg University Mainz, Mainz, Germany) was missing. This has been corrected in the HTML and PDF versions of the article. About this article Lang, F., Schrörs, B., Löwer, M. et al.
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Multiple instance learning to predict immune checkpoint blockade efficacy using neoantigen candidates

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By Patrick Sorn, Barbara Schrörs, David Weber, Franziska Lang
| BioRxiv
New Results doi: https://doi.org/10.1101/2022.05.06.490587 Abstract A successful response to immune checkpoint blockade treatment (ICB) depends on the functional re-invigoration of neoantigen-specific T cells and their anti-tumoral activity. Previous studies showed that the patient's neoantigen candidate load is an imperfect predictor of the response to ICB.
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