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Bart Theeuwes

As seen in: BioRxiv, Cell Press
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Articles

AXIN1 and AXIN2 regulate the WNT-signaling landscape to promote distinct mesoderm programs

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By Rocío Hernández-Martínez, Sonja Nowotschin, Luke Harland, Ying-Yi Kuo, Bart Theeuwes, Berthold Göttgens, Elizabeth H. Lacy, Anna-Katerina Hadjantonakis, Kathryn V. Anderson
| Cell Press Verified
STAR★Methods REAGENT or RESOURCESOURCEIDENTIFIER Antibodies Rat anti-CDH1 R&D systems Cat #MAB7481; RRID: AB_2076679 Rat anti-CDH1 Sigma-Aldrich Cat #U3254; RRID: AB_477600 Sheep anti-CDH2 R&D systems Cat #AF6426; RRID: AB_10718850 Rabbit anti-Brachyury (TBXT) Cell Signaling Technology Cat #81694; RRID: AB_2799983 Rabbit anti-CDX2 Cell Signaling Technology Cat #12306; RRID: AB_2797879 Goat anti-FOXF1 R&D systems Cat #AF4798; RRID: AB_2105588 Goat anti-TBX6 R&D systems Cat #AF4744; RRID:...
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Eomes directs the formation of spatially and functionally diverse extraembryonic hematovascular tissues

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By Bart Theeuwes, Luke Harland, Alexandra Bisia, Ita Costello, Mai-Linh N. Ton, Tim Lohoff, Stephen Clark, Ricard Argelaguet, Nicola Wilson, Wolf Reik, Elizabeth K. Bikoff, Elizabeth Robertson, Berthold Göttgens
| Cell Press Verified
Keywords embryogenesis extraembryonic mesoderm Eomes allantois yolk sac embryonic stem cell models single-cell analysis scRNA-seq scRNA + ATAC-seq endothelial development Introduction At the onset of murine gastrulation, extraembryonic mesodermal (ExEM) cells originating from the posterior region of the primitive streak (PS) migrate proximally to form hematovascular and mesenchymal cell types in both the visceral yolk sac (YS) and allantois (Figure 1A).
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STAT3 signalling enhances tissue expansion during postimplantation mouse development

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By Jenny Nichols, Takuya Azami, Bart Theeuwes, Mai-Linh N. Ton
| BioRxiv
Abstract STAT3 signalling has been studied extensively in the context of self-renewal and differentiation of mouse embryonic stem cells. Zygotic STAT3 is required for normal postimplantation development. On an outbred genetic background, Stat3 null embryos consistently lagged behind their littermates, beginning with significant reduction of epiblast cells at implantation. Remarkably, mutants closely resemble non-affected embryos from the previous day at all postimplantation stages examined.
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