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Benjamin L. Barthel on Muck Rack

Benjamin L. Barthel

As seen in: Color Research & Application, Wiley Online Library, The Journal of Clinical Investigation, Journal of Science and Medicine in Sport
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Articles

Universal Proteomic Signature After Exercise‐Induced Muscle Injury in Muscular Dystrophies

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By Benjamin L. Barthel, Mads G. Stemmerik, Nanna R. Andersen, Sofie V. Skriver
| Wiley Online Library Verified
1 Introduction Intense physical exercise, particularly activities with high eccentric load or exercise in unaccustomed individuals, results in muscle injury that is manifested by pain, swelling, reduced muscle strength and range of motion, and the release of muscle proteins such as creatine kinase (CK), titin, myoglobin (MYOG), and fast-myofiber troponin I (TNNI2) into the circulatory system [1-7].
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Elevation of fast but not slow troponin I in the circulation of patients with Becker and Duchenne muscular dystrophy

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By Benjamin L. Barthel, Dan Cox, Marissa Barbieri, Michael Ziemba
| Color Research & Application Verified
CLINICAL RESEARCH ARTICLE Alan J Russell PhD Corresponding Author arussell@edgewisetx.com orcid.org/0000-0001-7545-5008 Edgewise Therapeutics, BioFrontiers Institute, University of Colorado, Boulder, CO, USA Correspondence Alan J Russell, Edgewise Therapeutics, BioFrontiers Institute, University of Colorado, Boulder, CO 80303, USA. Email: arussell@edgewisetx.com.
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Large increases in plasma fast skeletal muscle troponin I after whole-body eccentric exercises

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By Trevor C. Chen, Hung-Wen Liu, Alan Russell, Benjamin L. Barthel
| Journal of Science and Medicine in Sport Verified
To view the full text, please login as a subscribed user or purchase a subscription. Click here to view the full text on ScienceDirect. Fig. 1 Enzyme-liked immunosorbent assay (ELISA) for human slow- (ssTnl) and fast-twitch skeletal troponin l (fsTnl) isoforms for muscle homogenates of biceps brachii (left) and soleus muscle (right) (A).
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