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Bethan Cole on Muck Rack

Bethan Cole

Shrewsbury
As seen in: Financial Times, MDPI, VICE, ELLE, British GQ, British Vogue, BioRxiv, Cell Press, Glamour (UK), Vogue Japan and
Writer and editor; cat, hedgehog, puffin and moomin lover. Contributing to Vogue Japan, Glamour, Red. Memoir (Everywhere is) Closed on Sundays - is on Amazon.
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Articles

August 2026 issue first authors

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By Bethan Cole, Hyemi Eo, Caiyiren Wen, Qingtong Zhou
| Cell Press Verified
TrendsTalk | Series: Meet the first authorsOnline now Affiliations & Notes 1Biochemical and Cellular Pharmacology, Genentech Inc., 103 DNA Way, South San Francisco, CA, 94080, United States of America 2Center for Regenerative Sciences, University of Texas Health Science Center San Antonio, San Antonio, TX, 78229, United States of America 3Zoology Department, Faculty of Science, Minia University, El-Minia, 61519, Egypt 4School of Life Science and Biotechnology, Kyungpook National University,...
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Structural pharmacology of TRPA1 channel

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By Bethan Cole, Huifen Chen, Alexis Rohou, Jun Chen
| Cell Press Verified
Keywords TRPA1 agonist antagonist cryo-EM binding sites drug discovery The emergence of TRPA1 as a therapeutic target The Transient Receptor Potential Ankyrin 1 (TRPA1) ion channel is the sole member of the mammalian TRPA subfamily. Predominantly expressed in nociceptive neurons (see Glossary) [1], TRPA1 functions as a polymodal receptor that integrates environmental irritants, oxidative insults, and cellular stress signals to regulate sensory signaling and neurogenic inflammation.
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Identification of new KCNT1-epilepsy drugs by in silico, cell and Drosophila modelling.

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By Michael G Ricos, Bethan Cole, Rashid Hussain, Grigori Rychkov
| BioRxiv
Abstract Objective: Hyperactive KCNT1 potassium channels, caused by gain-of-function mutations, are associated with a range of epilepsy disorders. Patients typically experience drug-resistant seizures and in cases with infantile onset, developmental regression can follow. KCNT1-related disorders include epilepsy of infancy with migrating focal seizures and sleep related hypermotor epilepsy.
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