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Brady Barron

As seen in: Nature
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Articles

Efferocytic remodelling of pancreatic islet macrophages by limited β-cell death - Nature

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By Brady Barron, Laurent Gorvel, Eynav Klechevsky, Xiaoxiao Wan
| Nature Verified
Abstract The primary driver of type I diabetes is the autoimmune T cells that destroy insulin-producing β-cells within the islets of Langerhans in the pancreas1. Pancreatic islet macrophages have also been variably linked to disease onset and progression. As macrophage-mediated removal of dying cells through efferocytosis regulates tissue homeostasis and immune responses2, here we investigated how efferocytosis by intra-islet macrophages influences the immune environment of pancreatic islets.
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Metabolites released from apoptotic cells act as tissue messengers - Nature

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By Sanja Arandjelovic, Brady Barron
| Nature Verified
Author notes These authors contributed equally: Parul Mehrotra, Sanja Arandjelovic, Justin Perry Authors and Affiliations Center for Cell Clearance, University of Virginia, Charlottesville, VA, USA Christopher B. Medina, Sanja Arandjelovic, Justin S. A. Perry, Sho Morioka, Brady Barron, Scott F. Walk & Kodi S. Ravichandran Department of Microbiology, Immunology, and Cancer Biology, University of Virginia, Charlottesville, VA, USA Christopher B. Medina, Sanja Arandjelovic, Justin S. A.
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Interpreting an apoptotic corpse as anti-inflammatory involves a chloride sensing pathway

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By Christopher Lucas, Kodi S. Ravichandran, Sho Morioka, Brady Barron
| Nature Verified
Abstract Apoptotic cell clearance (efferocytosis) elicits an anti-inflammatory response by phagocytes, but the mechanisms that underlie this response are still being defined. Here, we uncover a chloride-sensing signalling pathway that controls both the phagocyte ‘appetite’ and its anti-inflammatory response. Efferocytosis transcriptionally altered the genes that encode the solute carrier (SLC) proteins SLC12A2 and SLC12A4.
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