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Buddhiprabha Erabadda on Muck Rack

Buddhiprabha Erabadda

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Articles

Discovery of core genes for systemic lupus erythematosus via genome-wide aggregated trans-effects analysis - Genes & Immunity

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By Andrii Iakovliev, Olivia Castellini-Pérez, Buddhiprabha Erabadda, Javier Martin, Javier Martín, Guillermo Barturen, Paul M. McKeigue, Elena Carnero-Montoro, Marta Alarcon-Riquelme
| Nature Verified
Abstract The “omnigenic” hypothesis postulates that the polygenic effects of common variants on a typical complex trait coalesce on relatively few core genes through trans-effects on their expression. Our aim was to identify core genes for systemic lupus erythematosus (SLE) by testing for association with genome-wide aggregated trans-effects (GATE) scores for gene expression in a large genetic dataset (5267/4909 SLE cases/controls).
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Author Correction: Shared and disease-specific pathways in frontotemporal dementia and Alzheimer’s and Parkinson’s diseases - Nature Medicine

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By Muhammad Shoaib Ali, Buddhiprabha Erabadda, Yike Chen, Ying Xu, Katherine Gong, Menghan Liu, Daniel Western, Chengran Yang, Gyujin Heo, Jacob Vogel, Betty M. Tijms, Farhad Imam, Oskar Hansson
| Nature Verified
Correction to: Nature Medicine https://doi.org/10.1038/s41591-025-03833-1, published online 15 July 2025. This article was originally published under standard Springer Nature license (© The Author(s), under exclusive licence to Springer Nature America, Inc.). It is now available as an open-access paper under a Creative Commons Attribution 4.0 International license, © The Author(s). The error has been corrected in the HTML and PDF versions of the article.
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Shared and disease-specific pathways in frontotemporal dementia and Alzheimer’s and Parkinson’s diseases - Nature Medicine

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By Muhammad Shoaib Ali, Buddhiprabha Erabadda, Yike Chen, Ying Xu, Katherine Gong, Menghan Liu, Daniel Western, Chengran Yang, Gyujin Heo, Jacob Vogel, Betty M. Tijms, Farhad Imam, Oskar Hansson
| Nature Verified
Abstract Neurodegenerative diseases (NDs), such as Alzheimer’s disease (AD), Parkinson’s disease (PD) and frontotemporal dementia (FTD), exhibit distinct yet overlapping pathological mechanisms.
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