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Cedric Bardy on Muck Rack

Cedric Bardy

Australia, La Jolla
As seen in: Nature, Frontiers, Cell Press, Future Medicine
Professor and ARC Future Fellow at the South Australian Health and Medical Research Institute and Flinders University - Laboratory for Human Neurobiology
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Articles

Drug screen and machine learning predict neuroprotective agents in a preclinical human model of childhood dementia

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By Zarina Greenberg, Ella McDonald, Ella Mcdonald, Alejandra Noreña Puerta, Cade M. Christensen, Robert Adams, Jenne Tran, siti Mubarokah, Leanne Winner, Drew Neavin, Kristina L. Elvidge, Lisa Melton Verified, Mark Hutchinson Verified, Kim Hemsley, Nicholas Smith, Cedric Bardy, Manam Inushi De Silva, Paris Mazzachi, Sebastian Loskarn, Megan Maack
| Nature Verified
Abstract Childhood dementias are a group of paediatric neurodegenerative disorders characterised by neurocognitive decline, and in many cases underpinned by pathophysiological mechanisms similar to adult-onset dementias. In this study, we use patient-derived induced pluripotent stem cells (iPSCs) from children with one of the most prevalent childhood dementias, Mucopolysaccharidosis Type IIIA (MPS IIIA), also known as Sanfilippo syndrome.
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Repurposing trifluoperazine for glioblastoma treatment

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By Hui K. Gan, Cedric Bardy
| Cell Press Verified
Keywords brain cancer GBM glioma trifluoperazine antipsychotics NUPR1 ferroptosis dopamine calmodulin multidrug resistance chemotherapy phenothiazine Trifluoperazine as a novel therapeutic to target glioblastoma GBM is the most aggressive brain cancer in adults. Optimal treatment involves maximal safe resection, followed by irradiation and concomitant temozolomide (TMZ) chemotherapy [1]. However, prognosis is poor, with a median survival of 15 months and a 5 year survival rate of 10% [2].
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In vitro modeling of the human dopaminergic system using spatially arranged ventral midbrain–striatum–cortex assembloids - Nature Methods

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By Michael Zabolocki, Ilaria Morassut, Alessandro Fiorenzano, Molly Stevens, Cedric Bardy, Malin Parmar, Christian Krauditsch
| Nature Verified
Abstract Ventral midbrain dopaminergic neurons project to the striatum as well as the cortex and are involved in movement control and reward-related cognition. In Parkinson’s disease, nigrostriatal midbrain dopaminergic neurons degenerate and cause typical Parkinson’s disease motor-related impairments, while the dysfunction of mesocorticolimbic midbrain dopaminergic neurons is implicated in addiction and neuropsychiatric disorders.
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