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Chih-Ming Lin on Muck Rack

Chih-Ming Lin

As seen in: Nature, PLOS ONE, QJM, MDPI, Journal of Biomedical Science, Medscape, Royal Society of Chemistry
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Articles

Elevated trimethylamine levels characterize impaired muscle mass response to leucine-enriched protein supplementation in older adults at risk of sarcopenia

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By Hsiang-Yu Tang, Chi-Jen Lo, Hung-Yao Ho, Cheng-Hung Yang, Chen-Yu Chang, Yu-Hsuan Ho, Ji-Tseng Fang, Chih-Ming Lin, Mei-Ling Cheng
| Nature Verified
Abstract Leucine-enriched supplementation is a primary intervention for sarcopenia, yet individual responses vary. We integrated 1H-NMR metabolomics with clinical assessments in 47 older adults at high sarcopenia risk to identify metabotypes associated with improvements in muscle mass and strength. Following a 12-week intervention, distinct metabolic trajectories emerged between responders and non-responders.
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Accelerated peripheral nerve repair using surface-modified biomaterials for targeted capture of glial-derived growth factors post-neurorrhaphy

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By Shiu-Jau Chen, New Taipei, Chih-Ming Lin, Chiung-Hui Liu
| PLOS ONE
Loading metrics Open Access Peer-reviewed Research Article Citation: Chen S-J, Lin C-M, Liu C-H, Chen Y-H, Hsieh Y-L, Lin Y-C, et al. (2025) Accelerated peripheral nerve repair using surface-modified biomaterials for targeted capture of glial-derived growth factors post-neurorrhaphy. PLoS ONE 20(4): e0319979.
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An Approach for Personalized Dynamic Assessment of Chronic Kidney Disease Progression Using Joint Model

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By Chen-Mao Liao, Yi-Wei Kao, Yi-Ping Chang, Chih-Ming Lin
| MDPI
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
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