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Deeptankar DeMazumder on Muck Rack

Deeptankar DeMazumder

As seen in: National Institutes of Health, EP Europace, BioRxiv, eLife
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Articles

Ryanodine receptor 2 inhibition reduces dispersion of cardiac repolarization, improves contractile function, and prevents sudden arrhythmic death in failing hearts

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By Pooja Joshi, Shanea K. Estes, Deeptankar DeMazumder, Björn C. Knollmann
| eLife
All animal work followed IACUC-approved protocols at the respective institutions. A pressure overload model of HF and SCD was surgically generated with ascending aortic constriction (AC) and a daily bolus of low-dose isoproterenol (2 mg/kg/day) for β-adrenergic challenge. The surgical procedure and animal model have been previously described in detail (Dey et al., 2018). Briefly, animals were anesthetized with isoflurane then intubated.
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Ryanodine receptor 2 inhibition improves cardiac repolarization reserve and contractile function and prevents sudden arrhythmic death in failing hearts.

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By Pooja Joshi, Shanea K. Estes, Deeptankar DeMazumder, Björn C. Knollmann
| BioRxiv
Abstract Introduction: Sudden cardiac death (SCD) from ventricular tachycardia/fibrillation (VT/VF) are a leading cause of death, but current therapies are limited. Despite extensive research on drugs targeting sarcolemmal ion channels, none have proven sufficiently effective for preventing SCD. Sarcoplasmic ryanodine receptor 2 (RyR2) Ca2+ release channels, the downstream effectors of sarcolemmal ion channels, are underexplored in this context.
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Ryanodine receptor inhibition improves cardiac repolarization reserve and contractile function and prevents sudden arrhythmic death in failing hearts.

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By Pooja Joshi, Shanea K. Estes, Deeptankar DeMazumder, Björn C. Knollmann
| BioRxiv
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