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Articles by Dirk Strunk
DNA-binding affinity and specificity determine the phenotypic diversity in BCL11B-related disorders
Keywords BCL11B C2H2-type zinc finger protein recognition code genotype-phenotype correlation type 2 innate lymphoid cells Introduction Proteins containing a Cys2-His2 zinc-finger domain (C2H2-ZnF) constitute the largest class of human transcription factors.1 They act by either repressing or activating the expression of target genes through binding a specific DNA sequence by their tandem and modular C2H2-ZnFs. Each C2H2-ZnF shares a consensus sequence that folds in the presence of zinc (Zn2+)...
Author Correction: Sexually dimorphic estrogen sensing in skeletal stem cells controls skeletal regeneration - Nature Communications
Correction to: Nature Communications https://doi.org/10.1038/s41467-022-34063-5, published online 30 October 2022 In this article the author name Ruth Tevlin was incorrectly written as Ruth Telvin. The original article has been corrected.
Author Correction: Enhancer hijacking at the ARHGAP36 locus is associated with connective tissue to bone transformation - Nature Communications
Correction to: Nature Communications https://doi.org/10.1038/s41467-023-37585-8, published online 11 April 2023 In the Acknowledgements section of this article, one of the funding sources was incorrect.
Enhancer hijacking at the ARHGAP36 locus is associated with connective tissue to bone transformation - Nature Communications
Samples collection and ethics committee approval Healthy parents provided written informed consent on behalf of the female proband in this study. Blood collection for DNA screening and skin biopsy for fibroblast culture was performed after obtaining written consent from the parents. The study adhered to the Declaration of Helsinki standards and was approved by the internal Ethics Committee of the Department of Medical Sciences, University of Torino, Italy, under protocol number 0053916.
Sexually dimorphic estrogen sensing in skeletal stem cells controls skeletal regeneration - Nature Communications
Abstract Sexually dimorphic tissues are formed by cells that are regulated by sex hormones. While a number of systemic hormones and transcription factors are known to regulate proliferation and differentiation of osteoblasts and osteoclasts, the mechanisms that determine sexually dimorphic differences in bone regeneration are unclear. To explore how sex hormones regulate bone regeneration, we compared bone fracture repair between adult male and female mice.
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