Ece Canan Sayitoglu on Muck Rack

Ece Canan Sayitoglu

As seen in: Nature, Wiley Online Library, The BMJ
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Articles

379 Functional characterization of expanded HSC-derived NK cells, an optimal source for allogeneic cell therapy

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By Bauer L. LeSavage, Everardo Hegewisch-Solloa, Stefanie Maurer, Ece Canan Sayitoglu
| The BMJ Verified
Regular and Young Investigator Award Abstracts Cellular Therapies 379 Functional characterization of expanded HSC-derived NK cells, an optimal source for allogeneic cell therapy Statistics from Altmetric.com Request Permissions If you wish to reuse any or all of this article please use the link below which will take you to the Copyright Clearance Center’s RightsLink service. You will be able to get a quick price and instant permission to reuse the content in many different ways.
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AML/T cell interactomics uncover correlates of patient outcomes and the key role of ICAM1 in T cell killing of AML - Leukemia

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By Ece Canan Sayitoglu, Allison Boss, Molly Javier Uyeda, Yusuke Nakauchi, Norman J. Lacayo, Ravindra Majeti, Andrew J. Gentles, Alma-Martina Cepika
| Nature Verified
Abstract T cells are important for the control of acute myeloid leukemia (AML), a common and often deadly malignancy. We observed that some AML patient samples are resistant to killing by human-engineered cytotoxic CD4+ T cells. Single-cell RNA-seq of primary AML samples and CD4+ T cells before and after their interaction uncovered transcriptional programs that correlate with AML sensitivity or resistance to CD4+ T cell killing.
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AML/T cell interactomics uncover correlates of patient outcomes and the key role of ICAM1 in T cell killing of AML

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By Ece Canan Sayitoglu, Bogdan Luca, Allison Boss, Benjamin Thomas
| biorxiv.org
Abstract T cells are important for the control of acute myeloid leukemia (AML), a common and often deadly malignancy. We observed that some AML patient samples are resistant to killing by human engineered cytotoxic CD4+ T cells. Single-cell RNA-seq of primary AML samples and CD4+ T cells before and after their interaction uncovered transcriptional programs that correlate with AML sensitivity or resistance to CD4+ T cell killing.
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