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Elfriede Wieland

As seen in: Cell Press
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Articles

Endothelial Notch1 Activity Facilitates Metastasis

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By Elfriede Wieland, Juan Rodriguez-Vita, Sven S. Liebler, Carolin Mogler
| Cell Press Verified
Publication HistoryPublished: February 23, 2017Accepted: January 25, 2017; Received in revised form: September 28, 2016; Received: January 20, 2016;
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Cerebral Cavernous Malformation-1 Protein Controls DLL4-Notch3 Signaling Between the Endothelium and Pericytes

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By Gerald B. Schulz, Elfriede Wieland, Joycelyn Wüstehube-Lausch, Gwénola Boulday, Iris Moll, Elisabeth Tournier-Lasserve
| stroke.ahajournals.org
Background and Purpose—Cerebral cavernous malformation (CCM) is a neurovascular dysplasia characterized by conglomerates of enlarged endothelial channels in the central nervous system, which are almost devoid of pericytes or smooth muscle cells. This disease is caused by loss-of-function mutations in CCM1, CCM2, or CCM3 genes in endothelial cells, making blood vessels highly susceptible to angiogenic stimuli.
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