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Erica S Brady

As seen in: PLOS Biology, BioRxiv
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Articles

Hippocampal pyramidal cells of the CA1 region are not a major target of the thalamic nucleus reuniens

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By Lilya Andrianova, Paul Banks, Erica S Brady, Clair A. Booth
| PLOS Biology Verified
Loading metrics Open Access Peer-reviewed Research Article ? This is an uncorrected proof. Citation: Andrianova L, Banks PJ, Booth CA, Brady ES, Margetts-Smith G, Kohli S, et al. (2025) Hippocampal pyramidal cells of the CA1 region are not a major target of the thalamic nucleus reuniens. PLoS Biol 23(10): e3003419.
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Hippocampus does not appear to be a major target of thalamic nucleus reuniens

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By Lilya Andrianova, Erica S Brady, Gabriella Margetts-Smith, Shivali Kohli
| BioRxiv
Abstract The prefrontal - hippocampal - entorhinal system is perhaps the most widely-studied circuit in cognitive and systems neuroscience, due to its role in supporting cognitive functions such as working memory and decision-making. Disrupted communication within this circuit is a key feature of disorders such as schizophrenia and dementia. Nucleus reuniens (NRe) is a midline thalamic nucleus that sits at the nexus of this circuit, linking these regions together.
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Alterations to parvalbumin-expressing interneuron function and associated network oscillations in the hippocampal - medial prefrontal cortex circuit during natural sleep in APPNL-G-F mice

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By Jessica Griffiths, Lilya Andrianova, Takashi Saito, Erica S Brady
| BioRxiv
New Results doi: https://doi.org/10.1101/2022.02.08.479119 Abstract In the early stages of Alzheimer's disease (AD), the accumulation of the peptide amyloid-β (Aβ) damages synapses and disrupts neuronal activity and leads to disruption of neuronal oscillations associated with cognition. This is thought to be largely due to impairments in CNS synaptic inhibition, particularly via parvalbumin (PV)-expressing interneurons that essential for generating several key oscillations.
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