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Articles by Eviatar Fields
Reproducibility of Aquablation outcomes across surgeon experience levels: A descriptive analysis of published studies
Filename Description bco270282-sup-0001-Supplementary_Material.docxWord 2007 document , 22.4 KB Table S1: Reasoning for grouping of trials by surgeon experience. Japan [18], and Water I [20] and II [13] made up the early-experience group, while Water III[23], Mount Sinai[25], Madrid [16], ASC Pilot [24] and ASC Global [17] comprised the experienced group.
Elevator mechanism dynamics in a sodium-coupled dicarboxylate transporter
W. D. Stein, W. R. Lieb, Transport and Diffusion across Cell Membranes (Academic Press, 1986). L. R. Forrest, R. Krämer, C. Ziegler, The structural basis of secondary active transport mechanisms. Biochim. Biophys. Acta 1807, 167–188 (2011). D. Drew, O. Boudker, Shared molecular mechanisms of membrane transporters. Annu. Rev. Biochem. 85, 543–572 (2016). D. Markovich, “Sodium-sulfate/carboxylate cotransporters (SLC13)” in Current Topics in Membranes, M. O. Beven, Ed. (Elsevier, 2012), vol. 70, pp. 239–256. M. J.
Structured connectivity in the output of the cerebellar cortex - Nature Communications
Abstract The spatial organization of a neuronal circuit is critically important for its function since the location of neurons is often associated with function. In the cerebellum, the major output of the cerebellar cortex are synapses made from Purkinje cells onto neurons in the cerebellar nuclei, yet little has been known about the spatial organization of these synapses.
Structured connectivity in the output of the cerebellar cortex
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4E-BP2-dependent translation in cerebellar Purkinje cells controls spatial memory but not autism-like behaviors
Highlights • Ablation of 4E-BP2 in PCs (4E-BP2 cKO) impairs spatial memory and motor learning • Mice with deletion of 4E-BP2 in Purkinje cells (PCs) exhibit no autism-like phenotype • Increased regularity of action potential firing in 4E-BP2 cKO mice • Reduced number of Purkinje cells in mice lacking 4E-BP2 Summary Recent studies have demonstrated that selective activation of mammalian target of rapamycin complex 1 (mTORC1) in the cerebellum by deletion of the mTORC1 upstream repressors TSC1...
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