Felipe Batalini on Muck Rack

Felipe Batalini

Scottsdale
As seen in: Color Research & Application, Nature, Wiley, MDPI, Medscape, medRxiv, Chinese Clinical Oncology, European Journal of Cancer, Cancer Treatment Reviews
Physician-scientist, breast oncologist, genomics, bioinformatics. At Mayo Clinic & Harvard.
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Articles

Global Disparities in Artificial Intelligence-Based Mammogram Interpretation for Breast Cancer: A Scientometric Analysis of Representation, Trends, and Equity

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By Felipe Batalini, Leo A. Celi, Marisa Cobanaj
| European Journal of Cancer
Original ResearchArticles in Press115394Open access aDepartment of Medicine, Federal University of Parana, Curitiba, Parana, Brazil bDepartment of Radiology, Mayo Clinic, Phoenix, AZ, USA cSchool of Computing and Augmented Intelligence, Arizona State University, AZ, USA dArizona Advanced AI (A3I) Hub, Mayo Clinic, Phoenix, AZ, USA eDepartment of Oncology, Mayo Clinic, Phoenix, AZ, USA fDepartment of Radiology, University of Michigan, Ann Arbor, MI, USA gDepartment of Medicine, Beth Israel...
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Determinants of gastric cancer immune escape identified from non-coding immune-landscape quantitative trait loci - Nature Communications

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By Yered Pita-Juarez, Felipe Batalini, Shivani Nanda, Frank J. Slack, Xanthi-Lida Katopodi, Eleni Kanata, Nikolas Kalavros, Varune R. Ramnarine
| Nature
Abstract The landscape of non-coding mutations in cancer progression and immune evasion is largely unexplored. Here, we identify transcrptome-wide somatic and germline 3′ untranslated region (3′-UTR) variants from 375 gastric cancer patients from The Cancer Genome Atlas.
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Reciprocal antagonism of PIN1-APC/CCDH1 governs mitotic protein stability and cell cycle entry - Nature Communications

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By Fabin Dang, Lin Wang, Wenxue Li, Felipe Batalini, Jaymin Patel, John G. Clohessy, Yujing J. Heng, Yansheng Liu, Nathanael S. Gray, Gerburg Wulf
| Nature
Abstract Induced oncoproteins degradation provides an attractive anti-cancer modality. Activation of anaphase-promoting complex (APC/CCDH1) prevents cell-cycle entry by targeting crucial mitotic proteins for degradation. Phosphorylation of its co-activator CDH1 modulates the E3 ligase activity, but little is known about its regulation after phosphorylation and how to effectively harness APC/CCDH1 activity to treat cancer.
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