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Fiona Bartoli

As seen in: BioRxiv, The Journal of Biological Chemistry
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Articles

PIEZO1 force sensing controls global lipid homeostasis

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By Laeticia Lichtenstein, Elizabeth Evans, Hannah J Gaunt, Fiona Bartoli
| BioRxiv
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Orai1 Channel Inhibition Preserves Left Ventricular Systolic Function and Normal Ca2+ Handling After Pressure Overload

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By Fiona Bartoli, Marc Bailey, Baptiste Rode, Philippe Matéo
| AHA/ASA Journals
Originally published7 Jan 2020https://doi.org/10.1161/CIRCULATIONAHA.118.038891Circulation. ;0:null Abstract Background: Orai1 is a critical ion channel subunit, best recognized as a mediator of storeoperated Ca2+ entry (SOCE) in non-excitable cells. SOCE has recently emerged as a key contributor of cardiac hypertrophy and heart failure but the relevance of Orai1 is still unclear.
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Transient Receptor Potential Canonical (TRPC)/Orai1-dependent Store-operated Ca2+ Channels

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By Jessica Sabourin, Université Paris-Sud, Fiona Bartoli
| The Journal of Biological Chemistry Verified
Store-operated Ca2+ entry (SOCE) has emerged as an important mechanism in cardiac pathology. However, the signals that up-regulate SOCE in the heart remain unexplored. Clinical trials have emphasized the beneficial role of mineralocorticoid receptor (MR) signaling blockade in heart failure and associated arrhythmias. Accumulated evidence suggests that the mineralocorticoid hormone aldosterone, through activation of its receptor, MR, might be a key regulator of Ca2+ influx in cardiomyocytes.
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