Skip To Main Content
Gudny A. Arnadottir on Muck Rack

Gudny A. Arnadottir

As seen in: Nature
Is this you? As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work. Claim your profile

Get in touch with Gudny A.

Contact Gudny A., search articles and posts on X, monitor coverage, and track replies from one place.

Learn more about Muck Rack

Actions

Share this page

Is this you?

As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
Claim your profile

Articles

Sequence diversity lost in early pregnancy - Nature

|
By Gudny A. Arnadottir, Hakon Jonsson, Jennifer R. Gruhn, Peter Loof Møller, David Westergaard, Louise le Roux, Valgerdur Steinthorsdottir, Hannes P. Eggertsson, G. Bragi Walters, Hreinn Stefánsson, Agnar Helgason, Palle Duun Rohde, Bjarni V Halldorsson, Sofie Bliddal, Mette Nyegaard, Eva Hoffmann, Henriette Svarre Nielsen
| Nature Verified
Abstract Every generation, the human genome is shuffled during meiosis and a single fertilized egg gives rise to all of the cells of the body1. Meiotic errors leading to chromosomal abnormalities are known causes of pregnancy loss2,3, but genetic aetiologies of euploid pregnancy loss remain largely unexplained4.
Open in Who Shared Issue with article?

Rare loss-of-function variants in HECTD2 and AKAP11 confer risk of bipolar disorder - Nature Genetics

|
By Thorgeir E. Thorgeirsson, Vinicius Tragante, Gardar Sveinbjornsson, G. Bragi Walters, Erna V. Ivarsdottir, Gudny A. Arnadottir, Brynjar O. Jensson, Astros Th. Skuladottir, Gyda Bjornsdottir, Arni F. Gunnarsson, Rosa S. Gisladottir, Gudmar Thorleifsson, Engilbert Sigurdsson, Hilma Holm, Hreinn Stefánsson, Patrick Sulem
| Nature Verified
Abstract Bipolar disorder is a highly heritable psychiatric disorder; genome-wide association studies of bipolar disorder have yielded over 60 risk loci harboring common variants.
Open in Who Shared Issue with article?

Homozygosity for a stop-gain variant in CCDC201 causes primary ovarian insufficiency - Nature Genetics

|
By Asmundur Oddsson, Valgerdur Steinthorsdottir, Gudjon R. Oskarsson, Unnur Styrkarsdottir, Gísli H. Halldórsson, Gardar Sveinbjornsson, David Westergaard, Henriette Svarre Nielsen, Brynjar O. Jensson, Gudny A. Arnadottir, Hakon Jonsson, Ole A. Andreassen, G. Bragi Walters, Mette Nyegaard, Christian Erikstrup, Rolv T. Lie, Ingileif Jónsdóttir, Bjarni V Halldorsson, Gudmar Thorleifsson, Karina Banasik, Ole Birger Pedersen, Laufey Tryggvadottir, Jan Haavik, Hilma Holm, Thorunn Rafnar, Daniel F. Gudbjartsson, Patrick Sulem, Audunn S. Snaebjarnarson
| Nature Verified
Abstract Age at menopause (AOM) has a substantial impact on fertility and disease risk. While many loci with variants that associate with AOM have been identified through genome-wide association studies (GWAS) under an additive model, other genetic models are rarely considered1. Here through GWAS meta-analysis under the recessive model of 174,329 postmenopausal women from Iceland, Denmark, the United Kingdom (UK; UK Biobank) and Norway, we study low-frequency variants with a large effect on AOM.
Open in Who Shared Issue with article?
See all 10 articles