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Halla Elmore

As seen in: Nature, Cell Press
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Articles

The default mode network exhibits circuit-specific selective vulnerability despite widespread amyloid burden in an animal model of Alzheimer’s disease

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By Samuel J. Brunwasser, James McGregor, Kiran Bhaskaran-Nair, Clayton Farris, Halla Elmore, Bryan Higashikubo, Eva L Dyer, John Grady, Jennifer D. Whitesell, Julie Harris, Keith B. Hengen, Zachary D. Berriman-Rozen, Dongjun Ko
| Nature Verified
Abstract In Alzheimer’s Disease (AD) the default mode network (DMN) exhibits selective vulnerability to functional decline and amyloid beta (Aβ) plaques in early AD. DMN vulnerability may simply be the result of Aβ accumulation. Alternately, Aβ may unmask an intrinsic susceptibility of DMN circuits. Should DMN vulnerability persist amidst global Aβ deposition, this would support the latter hypothesis.
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