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Hamid Bolouri

As seen in: Nature, BioRxiv
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Articles

The Molecular Landscape of Pediatric Acute Myeloid Leukemia Reveals Recurrent Structural Alterations and Age-specific Mutational Interactions

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By Hamid Bolouri, Jason E Farrar, Timothy Triche, Rhonda Ries
| scholarsarchive.byu.edu
Pediatric acute myeloid leukemia, TARGET AML initiative, CpG methylation profiling Bolouri H, Farrar JE, Triche TJ, Ries RE, Lim EL, Alonzo TA, Ma Y, Moore R, Mungall AJ, Marra MA, Zhang J, Ma X, Liu Y, Liu Y, Auvil JMG, Davidsen TM, Gesuwan P, Hermida LC, Salhia B, Capone S, Ramsingh G, Zwaan CM, Noort S, Piccolo SR, Kolb EA, Gamis AS, Smith MA, Gerhard DS, and Meshinchi S.
Open in Who Shared Issue with article?

Unsupervised generative AI discovers pan-leukocyte dysregulated pathways in single-cell lupus data

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By Hamid Bolouri, Karen Cerosaletti, Adam Lacy-Hulbert
| BioRxiv
Abstract Comparisons of single-cell RNA-sequencing (scRNA-seq) data from healthy and diseased tissues are widely used to identify cell-type-specific dysregulated genes, pathways, and processes. Accordingly, many sophisticated methods have been developed for this purpose. However, such tools generally require considerable user expertise for optimal performance.
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Inflammatory bone marrow signaling in pediatric acute myeloid leukemia distinguishes patients with poor outcomes - Nature Communications

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By Hamid Bolouri, Rhonda Ries, Alice E Wiedeman, Vivian Gersuk, Jenny Smith, S. Alice Long, Sheila Scheiding
| Nature Verified
Abstract High levels of the inflammatory cytokine IL-6 in the bone marrow are associated with poor outcomes in pediatric acute myeloid leukemia (pAML), but its etiology remains unknown. Using RNA-seq data from pre-treatment bone marrows of 1489 children with pAML, we show that > 20% of patients have concurrent IL-6, IL-1, IFNα/β, and TNFα signaling activity and poorer outcomes.
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