Hayle Kincross on Muck Rack

Hayle Kincross

As seen in: The Journal of Clinical Investigation
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Articles

Loss of FBXO11 establishes a stem cell program in acute myeloid leukemia by dysregulating LONP1

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By Hayle Kincross, Xuan Wang, Linda Chang, Ya-Chi Angela Mo
| The Journal of Clinical Investigation
ResearchHematology Open Access | 10.1172/JCI181943 J Clin Invest. https://doi.org/10.1172/JCI181943. Copyright © 2025, Kincross et al. This work is licensed under the Creative Commons Attribution 4.0 International License. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. View PDF Abstract Acute myeloid leukemia (AML) is an aggressive cancer with very poor outcomes.
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Loss of FBXO11 function establishes a stem cell program in acute myeloid leukemia through dysregulation of the mitochondrial LONP1

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By Angela Mo, Xuan Wang, Linda Chang, Hayle Kincross
| biorxiv.org
Abstract Acute myeloid leukemia (AML) is an aggressive cancer with very poor outcomes. To identify additional drivers of leukemogenesis, we analyzed sequence data from 1,727 unique individual AML patients, which revealed mutations in ubiquitin ligase family genes in 11.2% of adult AML samples with mutual exclusivity. The Skp1/Cul1/Fbox (SCF) E3 ubiquitin ligase complex gene FBXO11 was the most significantly downregulated gene of the SCF complex in AML.
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