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Huanying Ge on Muck Rack

Huanying Ge

As seen in: Nature, Diabetes Magazine
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Articles

CRISPR−Cas9 CD33-deleted allogeneic hematopoietic cell transplantation with gemtuzumab ozogamicin maintenance in AML: a phase 1/2 trial

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By John DiPersio, Guenther Koehne, Nirali Shah, Lea BERNARD, Hyung C. Suh, Divya Koura, Roni Tamari, Muhammad Umair Mushtaq, Joseph E Maakaron, Joseph Cataquiz Rimando, Vanessa Kennedy, Sagar Patel Verified, Chad Hudson, Michael Loken, Deborah Lloyd, Darren Stanizzi, Melissa M. Lee-Sundlov, Sanjana A. Thosar, Guangwu Guo, Huanying Ge, Juliana Xavier-Ferrucio, Sharon Hyzy, Michelle Lin, Brenda Cooper, Christopher A. Slapak, Guy Mundelboim, Glen D. Raffel
| Nature Verified
Abstract Patients with high-risk acute myeloid leukemia (AML) and myelodysplastic syndrome (MDS) are likely to relapse despite allogenic hematopoietic cell transplantation (HCT). Post-HCT preventative maintenance can be limited by toxicity toward the normal donor cells.
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Haploinsufficiency of the Insulin Receptor in the Presence of a Splice-Site Mutation in Ppp2r2a Results in a Novel Digenic Mouse Model of Type 2 Diabetes

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By Ying Bai, Michelle Goldsworthy, Chi-Ming Li, Huanying Ge, Edwin Lamas
| Diabetes Magazine Verified
Insulin resistance in mice typically does not manifest as diabetes due to multiple compensatory mechanisms. Here, we present a novel digenic model of type 2 diabetes in mice heterozygous for a null allele of the insulin receptor and an N-ethyl-N-nitrosourea–induced alternative splice mutation in the regulatory protein phosphatase 2A (PP2A) subunit PPP2R2A. Inheritance of either allele independently results in insulin resistance but not overt diabetes.
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