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Huiying Zhao on Muck Rack

Huiying Zhao

As seen in: Nature, Wiley Online Library, Frontiers, MDPI, Taylor & Francis Online, Royal Society of Chemistry, BioRxiv, Cell Press, Dove Medical Press, Heart, Lung and Circulation and
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Articles

Slow-wave sleep engages brainstem circuitry to prevent stress-induced anxiety

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By Xiang Feng, Tao Le, Boyang Liu, Huiying Zhao, Yunting Su, Kaiyuan Li, Yujin Shao, Jinrong Liu, Zheyu Li, Guochuang Deng, Kelei Cao, Zhenggang Zhu, Jiadong Chen, Linghui Zeng, Yong Hee Han, Hongbin Yang, Yan-Qin Yu, Shumin Duan, Li Sun
| Cell Press Verified
Keywords slow-wave sleep anxiety stress parafacial zone lateral parabrachial nucleus oval bed nucleus of the stria terminalis Get full text access Log in, subscribe or purchase for full access. References 1. Scammell, T.E. ∙ Arrigoni, E. ∙ Lipton, J.O. Neural Circuitry of Wakefulness and Sleep Neuron. 2017; 93:747-765 2. Liu, D. ∙ Dan, Y. A Motor Theory of Sleep-Wake Control: Arousal-Action Circuit Annu. Rev. Neurosci. 2019; 42:27-46 3. Tovote, P. ∙ Fadok, J.P. ∙ Lüthi, A.
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A self-supervised electrocardiogram foundation model for empowering cardiovascular disease prediction and genetic factor discovery

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By Siying Lin, Zhaoqi Li, Qifan Wu, Yifeng Chen, Yuedong Yang, Huiying Zhao
| Nature Verified
Abstract Electrocardiogram (ECG) has been widely used in the diagnosis of cardiovascular disease (CVD). Current deep learning methods for CVD prediction using ECG often lack generalizability and interpretability, resulting in limited performance. Here, we have developed a self-supervised Electrocardiogram Large-scale Foundation Model (ECG-LFM) through pre-training over ten million 12-lead ECGs from multiple ECG datasets.
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Epigenome-wide Mendelian randomization with multi-omics validation identifies epigenetic drivers of idiopathic pulmonary fibrosis

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By Zhaoqi Li, Xingyu Lv, Shuangyu Yang, Linjie Huang, Huiying Zhao, Luna Hong, Fengzhan Ye, Shanping Jiang
| Nature Verified
Abstract Idiopathic pulmonary fibrosis (IPF) is a complex disease without clear etiology or effective therapy. While DNA methylation has been implicated in IPF pathogenesis, the tissue-specific causal effects of the epigenetic factors on IPF remain undetermined. Here, we perform epigenome-wide Mendelian randomization using blood-based methylation quantitative trait loci of 420,509 CpG sites and genome-wide association study for IPF to elucidate the causal effects of the CpG sites on IPF.
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