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Jennifer A. Barrie on Muck Rack

Jennifer A. Barrie

As seen in: Nature, JNeurosci
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Articles

TLR7-induced murine inflammation results in a global neuroinflammatory response driving neural circuit-specific transcriptomic changes

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By Lilya Andrianova, Rhona McGonigal, Deepika Sharma, Jennifer A. Barrie, Megan Saathoff, John Cole, Nigel Jamieson, Michael Craig, Jonathan Cavanagh, Kirstyn Gardner-Stephen, Robin J. Carvajal-Quisilema
| Nature Verified
Abstract Interactions between the brain and immune system play a key role in the aetiology of brain disorders, with inflammation emerging as a potential causal factor in subsets of major depressive disorder, particularly those resistant to treatment. The mechanisms through which immune activation might drive depressive symptoms remain elusive, limiting the ability to develop new targeted therapies.
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Schwann cell nodal membrane disruption triggers bystander axonal degeneration in a Guillain-Barré syndrome mouse model

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By Rhona McGonigal, Clare Campbell, Jennifer A. Barrie, Denggao Yao
| The Journal of Clinical Investigation
Abstract In Guillain-Barré syndrome (GBS), both axonal and demyelinating variants can be mediated by complement-fixing anti–GM1 ganglioside autoantibodies that target peripheral nerve axonal and Schwann cell (SC) membranes, respectively. Critically, the extent of axonal degeneration in both variants dictates long-term outcome. The differing pathomechanisms underlying direct axonal injury and the secondary bystander axonal degeneration following SC injury are unresolved.
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Glial Sulfatides and Neuronal Complex Gangliosides Are Functionally Interdependent in Maintaining Myelinating Axon Integrity

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By Mark McLaughlin, Rhona McGonigal, Jennifer A. Barrie, Denggao Yao
| JNeurosci Verified
Research Articles, Development/Plasticity/Repair Rhona McGonigal, Jennifer A. Barrie, Denggao Yao, Mark McLaughlin, Madeleine E. Cunningham, Edward G. Rowan and Hugh J. Willison Journal of Neuroscience 2 January 2019, 39 (1) 63-77; DOI: https://doi.org/10.1523/JNEUROSCI.2095-18.2018 Abstract Sulfatides and gangliosides are raft-associated glycolipids essential for maintaining myelinated nerve integrity.
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