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Jia En Wang

As seen in: Nature, Frontiers, Frontiers in Immunology, Frontiers in Public Health, MDPI, Canadian Journal of Infectious Diseases & Medical Microbiology, International Journal of Optics, Emerald Publishing, Monthly Weather Review, African Journal of Reproductive Health and

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Articles

The origin and domestication of modern cultivated Chrysanthemum × morifolium

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By Cunquan Yuan, Rui Zhang, Tianci Cong, Yalin Liu, Tingting Sun, Chunhua Li, Ming Sun, Qingqing Yang, Yujie Yang, Man Zhang, Yicen Yao, Qing Yuan, Xiaojie Song, Shuai Sun, Huitang Pan, Jia En Wang, Tangren Cheng, Tangchun Zheng, He Zhang, Guangyi Fan, Qixiang Zhang, Rouzhi Liu, Huien Zhao, Hetong Liu, Huiye Wu
| Nature Verified
Abstract The modern cultivated chrysanthemum (Chrysanthemum × morifolium Ramat.) is a globally important ornamental plant, ranking as the second-largest cut flower after roses. Here, to unravel its complex origin and domestication history, we achieved 6 haplotypes of the elite hexaploidy cut chrysanthemum cultivar ‘Jinba’ (2n = 6x = 54). By constructing a genome-wide variant map across 147 core accessions, we classified current Chrysanthemum varieties into 4 wild and 4 cultivated groups.
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Natural bioactive compounds targeting the macrophage clock in asthma: evidence, challenges, and perspectives

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By Baoyi Ni, Ru Chen, Zhu Liu, Xi Zhang, Huan He, Jia En Wang, Shaobin Sun
| Frontiers in Immunology
Abstract Background: Nocturnal asthma and airway remodeling are intimately linked to pulmonary circadian dysfunction. The dysregulation of core clock genes (e.g., Bmal1, Rev-erbα) in macrophages drives metabolic reprogramming and pro-fibrotic signaling. Pharmacological modulation of this clock using natural bioactive compounds offers a promising therapeutic strategy.
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Th17 cells and IL-17A in traumatic brain injury: mechanisms and therapeutic perspectives

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By Xin Guo, Jia En Wang, Siyu Zhu, Boyu Sun, Longlong Tian, Mingkang Li, Shiyao Feng, Guozhu Sun
| Frontiers in Immunology
Abstract Traumatic brain injury (TBI) triggers a complex cascade of immune responses that extend beyond the central nervous system and contribute to secondary neuropathology. Increasing evidence implicates T helper 17 cells (Th17) and their signature cytokine interleukin-17A (IL-17A) in post-traumatic neuroinflammatory processes, including blood-brain barrier (BBB) disruption and neuroimmune interactions.
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