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Jong Kim

As seen in: Nature, Wiley Online Library, MDPI, ACS Publications, BMC Cancer, Yomiuri Shimbun, Royal Society of Chemistry, BioRxiv, Canadian Journal of Infectious Diseases & Medical Microbiology, Journal of Applied Mathematics and

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Articles

Naturalistic adoption and deliberate practice use of an AI-based OSCE platform during national licensure preparation

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By Minseong Kim, Yoon Gyu Lee, Jong Kim, Junho Cho, young kim, Seng Chan You, Seung Won Cheong, Chaeryoung Park
| Nature Verified
Abstract AI-based tools for objective structured clinical examination (OSCE) preparation show promise under controlled evaluation, but whether learners voluntarily adopt them intensively during authentic high-stakes licensure preparation remains unclear.
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MIS-C Overlap, Not Prior COVID-19 Itself, Drives Treatment Escalation in Kawasaki Disease: A Nationwide Cohort Study

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By Min Kim, Jong Kim, Jihye You
| Wiley Online Library Verified
1 Introduction Kawasaki disease (KD) is an acute vasculitis that primarily affects children younger than 5 years and remains the leading cause of acquired heart disease in this age group [1-3]. Although its precise etiology remains unclear, KD is widely considered to reflect an abnormal immune response to infectious triggers in genetically susceptible children [4, 5].
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Intrinsic polyunsaturated fatty acid synthesis capacity dictates ferroptosis sensitivity under restricted arachidonic acid availability

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By Min Kim, Seo Young Jang, Ji-Yoon Lee, Jong Kim, Woosung Jung, Kyoung-Jin Oh, Baek-Soo Han, Kwang-Hee Bae, Mi-Young Kim, Geum-Sook Hwang, Eun-Woo Lee, Yeon-Mi You, Jooeun Kim
| Nature Verified
Abstract Ferroptosis is an iron-dependent type of regulated cell death driven by lipid peroxidation, in which polyunsaturated fatty acids (PUFAs) in membrane phospholipids serve as key substrates. Here, we identify PUFA biosynthetic capacity as a key determinant of ferroptosis sensitivity under arachidonic acid (AA)-limited conditions.
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