Jue Young Kim on Muck Rack

Jue Young Kim

As seen in: PLOS ONE
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Articles

Biobanking of gynecologic cancer biospecimens: Development, quality control, and translational applications

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By Jue Young Kim, Yonsei College, Yoo-kyung Lee
| PLOS ONE
Loading metrics Open Access Peer-reviewed Research Article Citation: Kim JY, Lee Y-K, Shin H-Y, Kim YJ, Jeon MA, Yang W, et al. (2026) Biobanking of gynecologic cancer biospecimens: Development, quality control, and translational applications. PLoS One 21(3): e0345861. https://doi.org/10.1371/journal.pone.0345861 Editor: Kazunori Nagasaka, Teikyo University, School of Medicine, JAPAN Received: September 23, 2025; Accepted: March 10, 2026; Published: March 31, 2026 Copyright: © 2026 Kim et al.
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Integrative Multi-Omics Analysis Reveals Molecular Signatures of Recurrence in Paired Primary and Recurrent High-Grade Serous Ovarian Cancer

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By Min-A Kim, Ha-Yeon Shin, Jue Young Kim, Johyeon Nam
| preprints.org
Submitted: 18 December 2025 You are already at the latest version High-grade serous ovarian cancer (HGSOC) is the most prevalent and aggressive form of epithelial ovarian cancer, characterized by high recurrence rates and poor clinical outcomes. In this study, we identify molecular signatures associated with recurrence by conducting integrative transcriptomic and proteomic analyses on paired primary and recurrent HGSOC tissues from 34 patients.
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PKM2 enhances cancer invasion via ETS-1-dependent induction of matrix metalloproteinase in oral squamous cell carcinoma cells

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By Xianglan Zhang, Young-Jin Park, Jue Young Kim, Doo Young Lee
| PLOS
Loading metrics Open Access Peer-reviewed Research Article Young-Jin Park , Jue Young Kim , Doo Young Lee , Xianglan Zhang, Shadavlonjid Bazarsad, Won-Yoon Chung, Jin Kim Young-Jin Park, Jue Young Kim, Doo Young Lee, Xianglan Zhang, Shadavlonjid Bazarsad, Won-Yoon Chung, Jin Kim Published: May 9, 2019 https://doi.org/10.1371/journal.pone.0216661 Figures Abstract Objectives This study aimed at investigating the molecular mechanism underlying PKM2-mediated cancer invasion.
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