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Jungsu Kim

As seen in: Medium, Nature, arXiv, Frontiers, MDPI, Cell Press
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Articles

Single-nucleus transcriptomics reveals disrupted pathways in the prefrontal cortex of Scn2a -deficient mice

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By Zaiyang Zhang, Purba Mandal, Zhengkuan Tang, Jingliang Zhang, Xiaoling Chen, Morgan Robinson, Muriel Eaton, Manasi Suchit Halurkar, Harish Kothandaraman, Luke C. Dabin, Boyu Jiang, Hongyu Gao, Chongli Yuan, Nadia A Lanman, Yunlong Liu, Jungsu Kim, Priyanka Baloni, Ye-Eun Yoo, Brody Deming
| Nature Verified
Abstract Loss-of-function variants in SCN2A, which encodes the NaV1.2 sodium channel critical for action potential initiation and backpropagation, are associated with autism spectrum disorder (ASD) and epilepsy. To investigate SCN2A deficiency–related phenotypes, we developed a preclinical mouse model with severe NaV1.2 deficiency, which exhibits robust behavioral abnormalities related to autism and epilepsy.
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Deletion of SPI1 in microglia exacerbates amyloid pathology by impairing microglial response in Alzheimer’s disease models

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By Byungwook Kim, Hande Karahan, H.R. Sagara Wijeratne, Ahmad Daniel Sharify, Selena Wang, Justin Kim, Mamun Al-Amin, Kelly Hartigan, Luke Child Dabin, Dominic J Acri, Emma H. Doud, Sutha K. John, Amber L. Mosley, Jungsu Kim, Mason Douglas Tate, Sunwoo Chung
| Cell Press Verified
Keywords Alzheimer’s disease amyloid amyloidosis microglia SPI1 PU.1 Syk Lyn Fcgr1 phagocytosis Introduction Alzheimer’s disease (AD) is a progressive neurodegenerative disorder histologically characterized by the accumulation of amyloid-beta (Aβ) plaques, neurofibrillary tangles, and dystrophic neurites.
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AVIN-Chat: An Audio-Visual Interactive Chatbot System with Emotional State Tuning

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By Seongmin Lee, Jungsu Kim, Jungbin Cho, Junwan Kim
| arXiv
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