Junho Kim on Muck Rack

Junho Kim

San Francisco
As seen in: Medium, Nature, Wiley Online Library, WIRED Magazine, MDPI, ACS Publications, American Chemical Society, Royal Society of Chemistry, Cell Press, Mechanical Engineering Magazine and
Creative Producer at Godfrey Dadich Partners | formerly WIRED
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Articles

Efficient, Patternable Full-Color Perovskite Quantum Dot LEDs via Defect-Passivating Film-State Ligand Engineering

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By Junho Kim, TaeHyun Kim, Seungjae Lee
| Wiley Online Library
Junho Kim, Taehyun Kim, Seungjae Lee and Seyun Lee contributed equally to this work. J.Kim, T.Kim, S.Lee, et al. “Efficient, Patternable Full-Color Perovskite Quantum Dot LEDs via Defect-Passivating Film-State Ligand Engineering.” Small (2026): e00011. https://doi.org/10.1002/smll.202600011 Conflicts of Interest The authors declare no conflicts of interest.
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Somatic cancer variants enriched in Alzheimer’s disease microglia-like cells drive inflammatory and proliferative states

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By August Yue Huang, Zinan Zhou, Maya Talukdar, Michael Miller, Brian Chhouk, Mengyue Zheng, Madel Durens, Minh T. Nguyen, Minh Nguyễn, Jaejoon Choi, Boxun Zhao, Sattar Khoshkhoo, Junho Kim, Rebecca B. Andersen, Zheming An, Yuchen Cheng, Javier Ganz, Levan Mekerishvili, Kyle J. Travaglini, Mariano I. Gabitto, Rebecca D. Hodge, Eitan S. Kaplan, Julia A. Belk, Dan A. Landau, Ed Lein, Philip De Jager, David Bennett, Samuele G. Marro, Eirini P. Papapetrou, Eunjung Lee, Christopher Walsh , Liz Enyenihi, Ila Rosen, Minye Zhou, Edward Stronge
| Cell Press
To examine the mutational burden of somatic variants in cancer driver genes in AD, we designed a hybrid capture gene panel covering 149 cancer driver genes with unique molecular identifier (UMI) barcoding (Table S1). We sequenced DNA from the prefrontal cortex (PFC) samples of 190 AD patients and 121 matched controls from the Religious Orders Study/Memory and Aging Project (ROSMAP) cohort at an average sequencing depth of >1,000× after UMI collapsing (Figures 1A, S1A, and S1B; Table S2).
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Somatic mosaicism in ALS and FTD identifies focal mutations associated with widespread degeneration

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By Zinan Zhou, Junho Kim, August Yue Huang, Matthew Nolan, Junseok Park, Ryan Doan, Taehwan Shin, Michael Miller, Boxun Zhao, Jinhyeong Kim, Katherine Morillo, Rebecca C. Yeh, Connor J. Kenny, Jennifer Neil, Takuya Ohkubo, John Ravits, Olaf Ansorge, Lyle W. Ostrow, Clotilde Lagier-Tourenne, Eunjung Lee, Christopher Walsh, Mingyun Bae, Brian Chhouk, Chao-Zong Lee
| Nature
Abstract Although mutations in many genes cause familial amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD), most cases are sporadic (sALS and sFTD) with unclear etiology. Here we tested whether somatic mutations contribute to sALS and sFTD by deep targeted sequencing of 88 neurodegeneration-related genes in postmortem brain and spinal cord samples from 399 sporadic cases and 144 controls.
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