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Kepeng Yan

As seen in: Nature, The Journal of Immunology
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Articles by Kepeng Yan

T-cell immunity in anti-tumor battle

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By Yingquan Qin, Kepeng Yan, Dongyang Li, Jinfeng Zhang, Yan Zhang, Wei Lu, Zongshuo Guo
| Nature Verified
Abstract The clinical success of T-cell-based immunotherapies has transformed oncology, yet tumor immune evasion remains a formidable barrier. In this review, we delineate the developmental and molecular landscapes that shape antitumor T-cell immunity. Following a brief overview of CD4+ helper T-cell contributions, we focus primarily on CD8+ cytotoxic lineages.
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Pathogenic Role of an IL-23/γδT17/Neutrophil Axis in Coxsackievirus B3-Induced Pancreatitis

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By Kepeng Yan, Jie Yang, Qian Qian, Dan Xu
| The Journal of Immunology Verified
Kepeng Yan, Jie Yang, Qian Qian, Dan Xu, Hui Liu, Lin Wei, Min Li and Wei Xu J Immunol December 15, 2019, 203 (12) 3301-3312; DOI: https://doi.org/10.4049/jimmunol.1900787 Key Points CVB3 infection induces a transient IL-17A expression in the pancreas of mice. Pancreatic IL-17A is mainly produced by Vγ4γδ T cells. IL-23/γδT17/neutrophil axis is critically involved in the onset of CVB3 pancreatitis. Abstract Coxsackievirus B is a common cause of viral myocarditis and pancreatitis.
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Pathogenic Role of an IL-23/γδT17/Neutrophil Axis in Coxsackievirus B3-Induced Pancreatitis

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By Jie Yang, Qian Qian, Dan Xu, Kepeng Yan
| The Journal of Immunology Verified
Kepeng Yan, Jie Yang, Qian Qian, Dan Xu, Hui Liu, Lin Wei, Min Li and Wei Xu J Immunol November 20, 2019, ji1900787; DOI: https://doi.org/10.4049/jimmunol.1900787 Key Points CVB3 infection induces a transient IL-17A expression in the pancreas of mice. Pancreatic IL-17A is mainly produced by Vγ4γδ T cells. IL-23/γδT17/neutrophil axis is critically involved in the onset of CVB3 pancreatitis. Abstract Coxsackievirus B is a common cause of viral myocarditis and pancreatitis.
Open in Who Shared Issue with article?
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