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Kimberley Anne Evans

As seen in: Nature, Cell Press
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Articles

Focal white matter lesions drive grey matter inflammation and synapse loss

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By Stavros Vagionitis, Andrea Lopez-López, Michael Perry, Bastien Herve, Balazs Varga, Eneritz Agirre, Sebastian Timmler, Mert Yucel, Kimberley Anne Evans, Yan Ting Ng, Courtney Kremler, Hélène Gautier, Yasmine Kamen, Helena Pivonkova, Katrin Volbracht, Javier Rueda-Carrasco, Soyon Hong, George G. Malliaras, Sabine Dietmann, Gonçalo Castelo-Branco, Ragnhildur Thora Karadottir, Omar de Faria Jr, Leslie Rodríguez-Kirby, Sabrina Ghosh, Andrew T. Setley, Tanja Mist Birgisdóttir, Sindri Gíslason, Felix Hildebrand, Christian A. Cepeda
| Nature Verified
Abstract Focal white matter lesions occur in most neurodegenerative disorders1,2,3. Despite occurring early in disease, white matter lesions are considered to be independent of, or secondary to, grey matter neuroinflammation, synapse loss and altered neuronal activity4,5,6,7. Notably, their functional effect on neuronal circuits remains understudied.
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Oligodendrocyte Progenitor Cells Become Regionally Diverse and Heterogeneous with Age

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By Sonia Spitzer, Sergey Sitnikov, Yasmine Kamen, Kimberley Anne Evans
| Cell Press Verified
Highlights • Oligodendrocyte progenitor cells (OPCs) acquire ion channels with age • OPCs become functionally heterogeneous both between brain regions and with age • NMDARs disappear in non-myelinating regions but remain in actively myelinating areas • Heterogeneity in OPC ion channel density indicates different functional states Summary Oligodendrocyte progenitor cells (OPCs), which differentiate into myelinating oligodendrocytes during CNS development, are the main proliferative cells in...
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