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Kunimichi Suzuki on Muck Rack

Kunimichi Suzuki

As seen in: Nature, Science Magazine, BioRxiv, Cell Press
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Articles

Structure and organization of AMPA receptor-TARP complexes in the mammalian cerebellum

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By Brian Cosgrove, Vincent Robert, Ksenia Krupina, Alexander Scrutton, Nayanika Sengupta, Josip Ivica, Imogen Stockwell, Sew Y. Peak-Chew, Bishal Singh, Kunimichi Suzuki, Veronica T. Chang, Stephen H. McLaughlin, James M. Krieger, A. Radu Aricescu, Ingo H. Greger
| Science Magazine Verified
Abstract AMPA receptors (AMPARs) are multimodal transducers of glutamatergic signals throughout the brain. Their diversity is exemplified in the cerebellum; at afferent synapses, AMPARs mediate high-frequency excitation, whereas in Bergmann glia (BG) they support calcium transients that modulate synaptic transmission. This spectrum arises from different combinations of core subunits (GluA1-4), auxiliary proteins, and post-transcriptional modifications.
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Operant House: A Versatile Open-Source Platform for Automated Operant Conditioning Testing of Mice in Home Cages

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By Shintaro Otsuka, Yuriko Nakamura, Aya Ito-Ishida, Kunimichi Suzuki
| BioRxiv
Abstract Operant conditioning is a valuable method for studying cognitive functions, yet its adoption is limited by low throughput, labor intensity, and high costs. Here, we developed Operant House, a low-cost, programmable device featuring a touchscreen, retractable levers, and a water reward port, designed for flexible, automated operant conditioning tasks.
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The complement C3-complement factor D-C3a receptor signalling axis regulates cardiac remodelling in right ventricular failure - Nature Communications

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By Hisayuki Hashimoto, Yohei Akiba, Kunimichi Suzuki, Manabu Abe, Yoshihiro Fukumoto, Keiichi Fukuda, Hirotaka Yada
| Nature Verified
Abstract Failure of the right ventricle plays a critical role in any type of heart failure. However, the mechanism remains unclear, and there is no specific therapy. Here, we show that the right ventricle predominantly expresses alternative complement pathway-related genes, including Cfd and C3aR1. Complement 3 (C3)-knockout attenuates right ventricular dysfunction and fibrosis in a mouse model of right ventricular failure.
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