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Lai Yee Cheong

As seen in: Nature, Wiley Online Library
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Articles

Author Correction: Adipose-targeted triiodothyronine therapy counteracts obesity-related metabolic complications and atherosclerosis with negligible side effects

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By Kang Chen, Lai Yee Cheong, Yuan Gao Verified, Yaming Zhang, Tianshi Feng, Qin Wang, Leigang Jin, Eric Honoré, Karen S. L. Lam, Weiping Wang, Xiaoyan Hui, Aimin Xu
| Nature Verified
Correction to: Nature Communications https://doi.org/10.1038/s41467-022-35470-4, published online 20 December 2022 In the version of this article initially published, due to a figure assembly error, in Fig. 6c, upper panel, the representative H&E staining image for LT3 was inadvertently chosen from the Saline group. This correction does not affect the study’s results or conclusions. The figure is now amended in the HTML and PDF versions of the article.
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Islet‐Resident Memory T Cells Orchestrate the Immunopathogenesis of Type 1 Diabetes through the FABP4‐CXCL10 Axis

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By Xiaoping Wu, Pharmacy The, Lai Yee Cheong, Lufengzi Yuan
| Wiley Online Library Verified
1 Introduction Type 1 diabetes (T1D), also known as autoimmune diabetes, is an organ-specific autoimmune disease precipitated by immune-associated destruction of insulin-producing pancreatic β cells.[1] T1D can lead to hyperglycemia-related microvascular complications and cardiovascular diseases, which primarily account for the shorter life expectancy among T1D patients.[2] However, there is currently no cure available for T1D. Most of the patients have to rely on lifelong insulin injections.
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Fibroblastic reticular cells in lymph node potentiate white adipose tissue beiging through neuro-immune crosstalk in male mice - Nature Communications

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By Lai Yee Cheong, Baile Wang, Qin Wang
| Nature Verified
Abstract Lymph nodes (LNs) are always embedded in the metabolically-active white adipose tissue (WAT), whereas their functional relationship remains obscure. Here, we identify fibroblastic reticular cells (FRCs) in inguinal LNs (iLNs) as a major source of IL-33 in mediating cold-induced beiging and thermogenesis of subcutaneous WAT (scWAT). Depletion of iLNs in male mice results in defective cold-induced beiging of scWAT.
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