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Leta Pilic on Muck Rack

Leta Pilic

As seen in: Nature, Memory Studies
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Articles

Interaction of melatonin receptor 1B ( MTNR1B ) genotype and type of breakfast (protein-enriched v carbohydrate-rich) on postprandial glucose response: a randomised crossover trial

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By Alexandra King, Jonathan Nixon, Angeliki Kapellou, Jonathan Craven, Catherine Graham, Leta Pilic, Yiannis Mavrommatis
| Nature Verified
Abstract The risk allele (G) of MTNR1B rs10830963 has been associated with impaired glucose tolerance, increased fasting glucose and type 2 diabetes (T2D). Late evening eating, when endogenous melatonin levels are elevated, is associated with impaired glucose control in MTNR1B risk carriers. Endogenous melatonin levels remain elevated into the morning so may influence glucose response to breakfast.
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Erythrocyte sodium buffering capacity status correlates with self-reported salt intake in a population from Livingstone, Zambia

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By Sepiso K. Masenga, Leta Pilic, St. Mary’s, Malani Malumani
| PLOS
Loading metrics Open Access Peer-reviewed Research Article Sepiso K. Masenga , Leta Pilic, Malani Malumani, Benson M. Hamooya Sepiso K. Masenga, Leta Pilic, Malani Malumani, Benson M. Hamooya Published: March 2, 2022 https://doi.org/10.1371/journal.pone.0264650 Figures Abstract Background Salt impairs endothelial function and increases arterial stiffness independent of blood pressure. The mechanisms are unknown.
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Does personalised nutrition advice based on apolipoprotein E and methylenetetrahydrofolate reductase genotype affect dietary behaviour?

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By Alexandra King, Jenna Smith Verified, Shaghayegh Saifi, Leta Pilic
| Memory Studies
Background Cardiovascular disease (CVD) is the most common cause of death worldwide, causing an estimated 17.9 million deaths globally (Wang et al., 2016). In the United Kingdom (UK) CVD is the second most common cause of death after cancer, causing approximately 168,000 deaths in 2017 (British Heart Foundation, 2019). CVD is a preventable cause of premature death and dietary intake is linked to numerous modifiable risk factors of CVD (NICE, 2010).
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