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Liya Yin on Muck Rack

Liya Yin

As seen in: Nature, Frontiers, International Journal of Cardiology
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Articles

Genetic or pharmacological inhibition of hepatic TMEM141 attenuates MASH and fibrosis via the ROS-HNF4α signaling pathway

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By Jiayou Wang, Chwen-Lih Chen, Raja Gopoju, Shuwei Hu, Yang Wang, Hui Wang, Xiaoli Pan, Yeong-Renn Chen, Jeffrey G. Mellott, Patrick Kang, Liya Yin, Yanqiao Zhang, Zaid Batayneh, Lakshitha Gunawardana
| Nature Verified
Abstract Metabolic dysfunction-associated steatoic liver disease (MASLD) and its advanced form, metabolic dysfunction-associated steatohepatitis (MASH), have become a global epidemic due to obesity and genetic susceptibility. Here, we report transmembrane protein 141 (TMEM141), a protein with a largely unknown function, as a key regulator of MASH and MASH-associated fibrosis. Hepatic TMEM141 is reduced in MASLD/MASH patients and mouse models of MASLD/MASH due to increased microRNA-149.
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Helicobacter pylori infection selectively attenuates endothelial function in male mice via exosomes-mediated ROS production

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By Veronica Folliero, Jiliang Zhou, Liya Yin
| Frontiers
Linfang Zhang1,2†, Xiujuan Xia1,2†, Hao Wu1, Xuanyou Liu1, Qiang Zhu1, Meifang Wang1, Hong Hao1, Yuqi Cui1, De-Pei Li1, Shi-You Chen3, Luis A. Martinez-Lemus1,4,5, Michael A.
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The role of MSC derived exosomes on cardiac microvascular dysfunction

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By Liya Yin, Vahagn Ohanyan, William M. Chilian, Feng Dong
| International Journal of Cardiology Verified
Liya Yin Vahagn Ohanyan William M. Chilian Feng Dong Feng Dong Correspondence Corresponding author at: Department of Integrative Medical Sciences, Northeast Ohio Medical University, 4209 State Route 44, P.O. Box 95, Rootstown, OH 44272, USA.
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