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Genotypic and Phenotypic Diversity as a Function of CRISPR Directed Gene Knock-Out of NRF2 in Pancreatic Adenocarcinoma Cells, a Feasibility Study

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By Kelly Banas, Gregory Tiesi, London McGill
| preprints.org
You are already at the latest version Pancreatic ductal adenocarcinoma (PDAC) presents unique treatment challenges often due to the development of anti-cancer drug resistance. Previously, we demonstrated that CRISPR-directed gene ablation disabled the master regulator gene NRF2, a tran-scription factor known to control drug resistance in squamous cell carcinoma tumor cells restored chemosensitivity.
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