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Marzia Del Re on Muck Rack

Marzia Del Re

Rome
As seen in: Nature, Wiley Online Library, MDPI, Future Medicine, European Urology, European Journal of Cancer, Lung Cancer, Cancer Treatment Reviews
Associate Professor Clinical Pharmacology @ Saint Camillus International University of Medical and Health Sciences | Fondazione Policlinico A. Gemelli IRCCS
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Articles

Dual actionability of BMI1 activation and mitotic vulnerability defines adaptive Osimertinib resistance in EGFR-mutant NSCLC

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By Paolo Armanetti, Maddalena Di Nardo, Chiara Cavallini, Stefania Crucitta, Marzia Del Re, Alice Chiodi, Ettore Mosca, Arianna Consiglio, Antonio Musio, Luca Menichetti, Emilia Bramanti, Azhar Ali, Elena Levantini, Giorgia Maroni, Raffaella Mercatelli, Nicola Losito
| Nature Verified
Abstract Non-small cell lung cancer (NSCLC) remains a leading cause of cancer mortality worldwide. Tumors carrying activating epidermal growth factor receptor (EGFR) mutations initially respond to tyrosine kinase inhibitors (TKIs), with Osimertinib representing the current standard of care. However, acquired resistance inevitably develops, involving both genetic and non-genetic mechanisms, the latter playing a major role in sustaining cellular plasticity and promoting tumor aggressiveness.
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HIF1A , ADRB3 , and VEGFA gene expression in human cord blood across gestation: insights toward a pharmacological artificial placenta

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By Rosa Teresa Scaramuzzo, Maura Calvani, Stefania Crucitta, Marzia Del Re, Riccardo Morganti, Maurizio Cammalleri, Paola Bagnoli, Alessandro Pini, Luca Filippi, Lucrezia Filippini, Bianca Tirinnanzi, Lorenza Di Marsico
| Nature Verified
Abstract Recent studies have shown that from the 23rd week of gestation onward, the fetus becomes progressively more hypoxic, with oxygenation levels rising again after 33–34 weeks. The biological significance of this biphasic oxygenation pattern has remained unclear. Umbilical cord blood samples from 100 preterm and 100 full-term neonates were analyzed for blood gas parameters and for HIF1A, ADRB3 (β3-adrenoceptor), and VEGFA gene expression.
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Covalent Bruton tyrosine kinase inhibitors across generations: A focus on zanubrutinib

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By Alessandro Broccoli, Marzia Del Re, Romano Danesi, Pier Luigi Zinzani
| Wiley Online Library Verified
1 INTRODUCTION Kinases are key regulatory components of the pathogenesis of several blood diseases. Targeting of tyrosine kinases with small molecules, given orally for an indefinite period of time, has become a well-established treatment strategy in chronic myeloid malignancies and—more recently—also in chronic B-cell lymphoproliferative diseases. The mechanism of kinase inhibition mainly rests on the binding of the inhibitor to the ATP-binding site of the enzyme.
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