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Max Benjamin Sauerland on Muck Rack

Max Benjamin Sauerland

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Articles

The binding of OTULIN restrains LUBAC activity to prevent TNF-driven immunopathology

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By Wenxin Lyu, Berthe Katrine Fiil, John Rizk, Majken Kjær, Max Benjamin Sauerland, Biao Ma, Malin Jessen, Hanna Kulvicki, Paul Elliott, Mads Gyrd-Hansen
| Cell Press Verified
Keywords OTULIN LUBAC PIM-PUB interaction LUBAC autoubiquitination Met1-Ub TNF signaling NF-κB activation cell death inflammation immune pathology Introduction Inflammation is an essential protective mechanism during infection but can cause tissue damage and fatality when dysregulated.1,2 Tumor necrosis factor (TNF) promotes inflammation through activation of mitogen-activated protein kinase (MAPK) and nuclear factor-kappa B (NF-κB) signaling and indirectly by inducing cell death.1,3 The...
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